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Volumn 20, Issue 4, 1999, Pages 609-617
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Chronic exposure to lead acetate affects the development of protein kinase C activity and the distribution of the PKCγ isozyme in the rat hippocampus
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Author keywords
Cognitive Development; Heavy Metal; Learning; Neurotoxicity
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Indexed keywords
LEAD ACETATE;
PROTEIN KINASE C;
ADOLESCENT;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BRAIN DEVELOPMENT;
COGNITIVE DEVELOPMENT;
CONTROLLED STUDY;
ENVIRONMENTAL EXPOSURE;
ENZYME ACTIVITY;
FETUS;
HIPPOCAMPUS;
LACTATION;
LEAD POISONING;
LEARNING;
NEUROTOXICITY;
NEWBORN;
NONHUMAN;
PRENATAL DRUG EXPOSURE;
PRIORITY JOURNAL;
RAT;
SYNAPTIC MEMBRANE;
AGE FACTORS;
ANIMALS;
ANIMALS, NEWBORN;
FEMALE;
FETUS;
HIPPOCAMPUS;
HISTONES;
IMMUNOBLOTTING;
ISOENZYMES;
LEAD;
MATERNAL-FETAL EXCHANGE;
ORGANOMETALLIC COMPOUNDS;
PHOSPHOLIPIDS;
PREGNANCY;
PROTEIN KINASE C;
RATS;
RATS, SPRAGUE-DAWLEY;
SYNAPSES;
TIME FACTORS;
ANIMALIA;
HIPPOCAMPUS HIPPOCAMPUS;
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EID: 0032873525
PISSN: 0161813X
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
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Times cited : (24)
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References (56)
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