Size does matter: Will knockout of p21(WAF1/CIP1) save the kidney by limiting compensatory renal growth?

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 19, 1999, Pages 10551-10553

  Al Awqati, Q ^a   Preisig, P A ^a  

^a New York Presbyterian Columbia University Medical Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN P21; TRANSFORMING GROWTH FACTOR BETA;

ANIMAL EXPERIMENT; ANIMAL MODEL; GENE EXPRESSION REGULATION; GLOMERULUS FILTRATION RATE; GROWTH REGULATION; HOMEOSTASIS; HYPERPLASIA; KIDNEY DEVELOPMENT; KIDNEY FAILURE; KIDNEY HYPERTROPHY; KNOCKOUT GENE; MOUSE; NONHUMAN; NOTE; PRIORITY JOURNAL; UREMIA;

ANIMALS; BODY WEIGHT; CELL CYCLE; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLIN-DEPENDENT KINASES; CYCLINS; DIET THERAPY; HYPERPLASIA; HYPERTROPHY; KIDNEY; KIDNEY FAILURE; MICE; MICE, KNOCKOUT; NEPHRONS; TRANSFORMING GROWTH FACTOR BETA; UREMIA;

ANIMALIA; GLOMERULUS;

EID: 0032843760     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.19.10551     Document Type: Note

References (17)