Can we learn from the pathogenetic strategies of group A hemolytic streptococci how tissues are injured and organs fail in post-infectious and inflammatory sequelae?

FEMS Immunology and Medical Microbiology

Volumn 25, Issue 4, 1999, Pages 325-338

  Ginsburg, Isaac ^a   Ward, Peter A ^b   Varani, James ^b  

^a HEBREW UNIVERSITY OF JERUSALEM   (Israel)

^b UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

Author keywords

Lysosomal hydrolase; Post infectious sequela; Sepsis; Streptococcus; Synergism; Toxin

Indexed keywords

ANTIBIOTIC AGENT; ANTIINFECTIVE AGENT; ARACHIDONIC ACID; CATHEPSIN; CATHEPSIN G; CHEMOKINE; COMPLEMENT; CYTOKINE; DEFENSIN; ELASTASE; HEMOLYSIN; LEUKOCYTE ELASTASE; LYSOSOME ENZYME; MONOCLONAL ANTIBODY; NITROGEN OXIDE; NONSTEROID ANTIINFLAMMATORY AGENT; OXIDIZING AGENT; PENTOXIFYLLINE; PHOSPHOLIPASE; PHOSPHOLIPASE C; PLASMIN; PROTEIN TYROSINE KINASE INHIBITOR; PROTEINASE; SERINE PROTEINASE; STREPTOLYSIN O; STREPTOLYSIN S; SUPEROXIDE; TRYPSIN;

APOPTOSIS; BACTERIAL INFECTION; BACTERIOLYSIS; CELL DAMAGE; HUMAN; INFLAMMATION; NONHUMAN; PATHOGENESIS; PATHOPHYSIOLOGY; PHAGOCYTOSIS; PRIORITY JOURNAL; REVIEW; SEPSIS; SEPTIC SHOCK; STREPTOCOCCUS GROUP A; TISSUE INJURY;

EID: 0032790246     PISSN: 09288244     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0928-8244(99)00107-8     Document Type: Article

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