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Electrophysiological study of neuromuscular blocking action of cobra neurotoxin
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Influence of chronic neostigmine treatment on the number of acetylcholine receptor and the release of acetylcholine of the rat diaphragm
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Differentiation between intrinsic and extrinsic acetylcholine receptors of the chick biventer cervicis muscle
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Turnover of junctional and extrajunctional acetylcholine receptors of the rat diaphragm
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Chang, CC, Chuang ST, Huang MC. Effects of chronic treatment with various neuromuscular blocking agents on the number and distribution of acetylcholine receptors in the rat diaphragm. J Physiol (Lond) 250;161-173;1975.
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Further evidence that extrinsic acetylcholine acts preferentially on extrajunctional receptors in the chick biventer cervicis muscle
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Chang CC, Su MJ. Further evidence that extrinsic acetylcholine acts preferentially on extrajunctional receptors in the chick biventer cervicis muscle. Eur J Pharmacol 33:337-344; 1975.
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A study on the membrane depolarization of skeletal muscles caused by a scorpion toxin, sea anemone toxin II and crotamine and the interaction between toxins
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Chang CC, Hong SJ, Su MJ. A study on the membrane depolarization of skeletal muscles caused by a scorpion toxin, sea anemone toxin II and crotamine and the interaction between toxins. Br J Pharmacol 79;673-680;1983.
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A regenerative release of acetylcholine from mouse motor nerve terminals treated with anticholinesterase agents
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Dissociation of the endplate potential run down and the tetanic fade from the postsynaptic inhibition of acetylcholine receptor by α-neurotoxins
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Reversals of the neostigmine-induced tetanic fade and endplate potential run-down with respect to the autoregulation of transmitter release
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Chang CC, Chen SM, Hong SJ. Reversals of the neostigmine-induced tetanic fade and endplate potential run-down with respect to the autoregulation of transmitter release. Br J Pharmacol 95:1255-1261;1988.
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Feedback modulation of acetylcholine release from motor nerve
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Chang CC, Chiou LC, Hwang LL. Selective anlagonism to succinylcholine-induced depolarization by α-hungarotoxin with respect to the mode of action of depolarizing agents. Br J Pharmacol 98:1413-1419;1989.
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Use of a snake venom toxin to characterize the cholinergic receptor protein
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