Defective processing and expression of thiazide-sensitive Na-Cl cotransporter as a cause of Gitelman's syndrome

American Journal of Physiology - Renal Physiology

Volumn 277, Issue 4 46-4, 1999, Pages

  Kunchaparty, Shanti ^b   Palcso, Matthew ^a   Berkman, Jennifer ^a   Velázquez, Heino ^b   Desir, Gary V ^b   Bernstein, Paul ^b   Reilly, Robert F ^a   Ellison, David H ^a,c  

^a UNIVERSITY OF COLORADO   (United States)

^b YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c Children's Medical Center   (United States)

Author keywords

Blood pressure; Distal; Distal convoluted tubule; Diuretics; Familial hypokalemia hypomagnesemia; Kidney tubules; Thiazide; Thiazide sensitive sodium chloride cotransporter

Indexed keywords

THIAZIDE DIURETIC AGENT;

ANIMAL CELL; ARTICLE; CHLORIDE TRANSPORT; GITELMAN SYNDROME; HYPOKALEMIA; HYPOMAGNESEMIA; ION EXCHANGE; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN PROCESSING; SODIUM TRANSPORT;

EID: 0032698959     PISSN: 1931857X     EISSN: 15221466     Source Type: Journal    
DOI: 10.1152/ajprenal.1999.277.4.f643     Document Type: Article

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