In cortical cultures of trisomy 16 mouse brain the upregulated metallothionein-I/II fails to respond to H2O2 exposure or glutamate receptor stimulation

Brain Research

Volumn 787, Issue 2, 1998, Pages 292-298

  Scortegagna, Marzia ^a   Galdzicki, Zygmunt ^b   Rapoport, Stanley I ^b   Hanbauer, Ingeborg ^a  

^a NATIONAL HEART LUNG AND BLOOD INSTITUTE   (United States)

^b NATIONAL INSTITUTE ON AGING   (United States)

Author keywords

Cortical primary culture; Glutamate receptor stimulation; Hydrogen peroxide; Metallothionein I II; Oxidative stress; Trisomy 16 mouse; Western blot analysis

Indexed keywords

ACETYLCYSTEINE; GLUTAMATE RECEPTOR AGONIST; HYDROGEN PEROXIDE; KAINIC ACID; METALLOTHIONEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; CHROMOSOME 16; CONTROLLED STUDY; FEMALE; FETUS; MALE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN CONTENT; TRISOMY;

ACETYLCYSTEINE; ANIMALS; ASTROCYTES; BLOTTING, WESTERN; CEREBRAL CORTEX; EXCITATORY AMINO ACID AGONISTS; FEMALE; FREE RADICAL SCAVENGERS; HYDROGEN PEROXIDE; LEAD; MALE; METALLOTHIONEIN; MICE; MICE, INBRED C57BL; OXIDANTS; OXIDATION-REDUCTION; RECEPTORS, GLUTAMATE; STIMULATION, CHEMICAL; TRISOMY; UP-REGULATION;

EID: 0032559868     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-8993(97)01501-1     Document Type: Article

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