Repression of B7.2 on self-reactive B cells is essential to prevent proliferation and allow Fas-mediated deletion by CD4+ T cells

Journal of Experimental Medicine

Volumn 188, Issue 4, 1998, Pages 651-659

  Rathmell, Jeffrey C ^a,d   Fournier, Sylvie ^c   Weintraub, Bennett C ^a,b   Allison, James P ^c   Goodnow, Christopher C ^a,b  

^a STANFORD UNIVERSITY   (United States)

^b Biology and Environment   (Australia)

^c UNIVERSITY OF CALIFORNIA   (United States)

^d UNIVERSITY OF CHICAGO   (United States)

Author keywords

Autoimmunity; B cell; B cell antigen receptor; B7.2 (CD86); Fas (CD95)

Indexed keywords

CD4 ANTIGEN; CD86 ANTIGEN; FAS ANTIGEN; FAS LIGAND; LYMPHOCYTE ANTIGEN RECEPTOR;

ADOPTIVE TRANSFER; ANIMAL CELL; ARTICLE; AUTOIMMUNITY; B LYMPHOCYTE; CONTROLLED STUDY; ENZYME LINKED IMMUNOSORBENT ASSAY; FLOW CYTOMETRY; LYMPHOCYTE PROLIFERATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; T LYMPHOCYTE;

ANIMALS; ANTIGENS, CD; ANTIGENS, CD27; ANTIGENS, CD86; ANTIGENS, CD95; B-LYMPHOCYTES; CD4-POSITIVE T-LYMPHOCYTES; CELL DIFFERENTIATION; CELL DIVISION; CLONAL ANERGY; FAS LIGAND PROTEIN; FEMALE; GENE EXPRESSION REGULATION; INTERLEUKIN-2; INTERLEUKIN-4; MALE; MEMBRANE GLYCOPROTEINS; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MURAMIDASE; RECEPTORS, OX40; RECEPTORS, TUMOR NECROSIS FACTOR; TRANSGENES;

EID: 0032541486     PISSN: 00221007     EISSN: None     Source Type: Journal    
DOI: 10.1084/jem.188.4.651     Document Type: Article

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