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Volumn 95, Issue 8, 1998, Pages 4350-4355

Human PEX1 cloned by functional complementation on a CHO cell mutant is responsible for peroxisome-deficient Zellweger syndrome of complementation group I

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL CELL; ARTICLE; CHO CELL; CONTROLLED STUDY; GENETIC COMPLEMENTATION; HUMAN; HUMAN CELL; MOLECULAR CLONING; NONHUMAN; NUCLEOTIDE SEQUENCE; PEROXISOME; POINT MUTATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; ZELLWEGER SYNDROME;

EID: 0032515992     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.95.8.4350     Document Type: Article
Times cited : (87)

References (48)
  • 4
    • 0000228425 scopus 로고
    • eds. Scriver, C. R., Beaudet, A. I., Sly, W. S. & Valle, D. (McGraw-Hill, New York), 7th Ed.
    • Lazarow, P. B. & Moser, H. W. (1995) in The Metabolic Basis of Inherited Disease, eds. Scriver, C. R., Beaudet, A. I., Sly, W. S. & Valle, D. (McGraw-Hill, New York), 7th Ed., pp. 2287-2324.
    • (1995) The Metabolic Basis of Inherited Disease , pp. 2287-2324
    • Lazarow, P.B.1    Moser, H.W.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.