Abrogation of the G2 cell cycle checkpoint associated with overexpression of HSIX1: A possible mechanism of breast carcinogenesis

Proceedings of the National Academy of Sciences of the United States of America

Volumn 95, Issue 21, 1998, Pages 12608-12613

  Ford, Heide L ^a,c   Kabingu, Edith N ^a   Bump, Edward A ^a   Mutter, George L ^b   Pardee, Arthur B ^a  

^a HARVARD MEDICAL SCHOOL   (United States)

^b BRIGHAM AND WOMEN S HOSPITAL   (United States)

^c DANA FARBER CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BREAST CANCER; BREAST CARCINOGENESIS; CELL CYCLE G2 PHASE; CELL CYCLE S PHASE; CONFERENCE PAPER; CONTROLLED STUDY; FLUORESCENCE ACTIVATED CELL SORTER; HOMEOBOX; HUMAN; HUMAN CELL; MOLECULAR CLONING; PRIORITY JOURNAL; PROTEIN EXPRESSION; ARTICLE; BREAST TUMOR; CELL CULTURE; CELL TRANSFORMATION; GENE; GENETICS; NUCLEOTIDE SEQUENCE; PATHOLOGY;

PRIMER DNA;

BASE SEQUENCE; BREAST NEOPLASMS; CELL TRANSFORMATION, NEOPLASTIC; DNA PRIMERS; G2 PHASE; GENES, CDC; GENES, HOMEOBOX; HUMANS; TUMOR CELLS, CULTURED;

EID: 0032514597     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.95.21.12608     Document Type: Article

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