Angioneurotic oedema due to C1-Esterase inhibitor deficiency;Angioodeme durch C1-Esterase-Inhibitor-Mangel

Deutsche Medizinische Wochenschrift

Volumn 123, Issue 23, 1998, Pages 737-740

  Jentsch Ullrich, K ^a   Leuner, S ^a   Kahl, Ch ^a   Arland, M ^a   Florschutz A ^a   Franke, A ^a   Hoffkes H G ^a  

^a OTTO VON GUERICKE UNIVERSITY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

AMINOCAPROIC ACID; ANDROGEN; APROTININ; BERINERT HS; COMPLEMENT COMPONENT C1S INHIBITOR; CYCLOPHOSPHAMIDE; DANAZOL; FRESH FROZEN PLASMA; IMMUNOGLOBULIN; IMMUNOSUPPRESSIVE AGENT; STANOZOLOL; TRANEXAMIC ACID; UNCLASSIFIED DRUG; ANABOLIC AGENT; ANTIESTROGEN; ANTIFIBRINOLYTIC AGENT;

ANGIONEUROTIC EDEMA; CANCER; HUMAN; INTRAVENOUS DRUG ADMINISTRATION; MUSCLE NECROSIS; MYOSITIS; ORAL DRUG ADMINISTRATION; PATHOGENESIS; PLASMAPHERESIS; PROPHYLAXIS; SHORT SURVEY; SYMPTOMATOLOGY; THROMBOSIS; PHYSIOLOGY; REVIEW;

ANABOLIC AGENTS; ANGIONEUROTIC EDEMA; ANTIFIBRINOLYTIC AGENTS; COMPLEMENT C1 INACTIVATOR PROTEINS; DANAZOL; ESTROGEN ANTAGONISTS; HUMANS; IMMUNOSUPPRESSIVE AGENTS; PLASMAPHERESIS; STANOZOLOL;

EID: 0032485978     PISSN: 00120472     EISSN: None     Source Type: Journal    
DOI: 10.1055/s-2007-1024047     Document Type: Short Survey

References (15)

1. Agostoni, A., M. Cicardi: Replacement therapy in hereditary and acquired angioedema. Pharm. Res. 26 (1992), 148-149.
2. Arreaza, E. E., K. Singh, A. Grant: Hereditary angioedema: clinical and biochemical heterogenity. Ann. Allergy 61 (1988), 69-75.
3. Bain, B. J., D. Catovsky, P. W. Ewan: Acquired angioedema as the presenting feature of lymphoproliferative disorders of mature B lymphocytes. Cancer 72 (1993), 3318-3322.
4. Cohen, S. H., S. M. Koethe, F. Kozin, G. Rodey, J. A. Arkins, J. N. Fink: Acquired angioedema associated with rectal carcinoma and its respond to danazol therapy. J. Allergy Clin. Immunol. 62 (1978), 217-221.
5. Donaldson, V. H., O. D. Ratnoll, U. P. Da Silva, F. S. Rosen: Permeability-increasing activity in hereditary angioneurotic edema plasma. J. Clin. Invest. 48 (1969), 642-670.
6. Donaldson, V. H., D. I. Bernstein, C. J. Wagner, B. H. Mitchell, J. Scinto, I. L. Bernstein: Angioneurotic edema with acquired C1 inhibitor deficiency and autoantibody to C1 inhibitor. Response to plasmapheresis and cytotoxic therapy. J. Lab. Clin. Med. 119 (1992), 397-406.
7. Frank, M. M., A. Malbran, H. Simms, K. Melez, U. Santaella, C. Hammer, L. Fries: Acquired angioedema type II. A new autoimmune disease. Clin. Research. 35 (1987), 641 A.
8. Gadek, J. E., J. D. Fulmer, J. A. Gelfand, M. M. Frank, T. L. Petty, R. G. Crystal: Danazol-induced augmentation of serum 1-antitrypsin levels in individuals with marked deficiency of this antiprotease. J. Clin. Invest. 66 (1980), 82-87.
9. Grace, R. J., A. Jacob, C. J. Mainwaring, B. A. Mc Verry: Acquired C1 esterase inhibitor deficiency as manifestation of T-cell lymphoproliferative disorder. Lancet 336 (1990), 118.
10. Gelfand, J. A., G. R. Boss, C. L. Conley, R. Reinhart, M. M. Frank: Acquired C1 esterase inhibitor deficiency and angioedema. A review. Medicine 58 (1979), 321-328.
11. Hauptmann, G., F. Petitjean, J. M Lang, F. Oberling: Acquired C1-inhibitor deficiency in a case of lymphosarcoma of the spleen. Reversal of complement abnormalities after splenectomy. Clin. Exp. Immunol. 37 (1979), 523-531.
12. Nilson, A., R. Matre: Acquired angioedema and hypocomplementemia in a patient with myelofibrosis. Effect of danazol treatment. Acta Med. Scand. 207 (1980): 123-125.
13. Opferkuch, W., K. Echternacht, W. Gronemeyer, C. Hammar, U. Jaeger, H. Niemczyk, C. Rieger: Klinische Ausprägung und Therapie des hereditären angioneurotischen Ödems (HANE). Immun. Infekt. 8 (1981), 56-60.
14. Sheffer, A. L., K. F. Austen, F. S. Rosen, D. T. Fearin: Acquired deficiency of the inhibitor of the first component of complement. Report of five additional cases with commentary on the syndrome. J. Allergy Clin. Immunol. 75 (1985), 640-646.
15. Späth, P. J., B. Wüthrich, R. Bütler: Quantification of C1-inhibitor functional activity by immunodiffusion assay in plasma of patients with hereditary angioedema - evidence of a functionally critical level of C1-INH-concentrations. Complement 1 (1984), 147-159.