CA1 long-term potentiation is diminished but present in hippocampal slices from α-CaMKII mutant mice

Learning and Memory

Volumn 5, Issue 4-5, 1998, Pages 344-354

  Hinds, Heather L ^a,b   Tonegawa, Susumu ^b   Malinow, Roberto ^a  

^a Simons Center for Quantitative Biology   (United States)

^b HOWARD HUGHES MEDICAL INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ISOENZYME; PROTEIN KINASE (CALCIUM,CALMODULIN) II;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME DEFECT; ENZYME POLYMORPHISM; EXCITATORY POSTSYNAPTIC POTENTIAL; HIPPOCAMPUS; LONG TERM POTENTIATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; SYNAPTIC TRANSMISSION;

ANIMALS; CA(2+)-CALMODULIN DEPENDENT PROTEIN KINASE; CROSSES, GENETIC; ELECTRIC STIMULATION; FEMALE; HIPPOCAMPUS; LONG-TERM POTENTIATION; MALE; MICE; MICE, INBRED BALB C; MICE, INBRED C57BL; MICE, INBRED STRAINS; MICE, KNOCKOUT; NEURONS; REGRESSION ANALYSIS; SYNAPSES;

EID: 0032456783     PISSN: 10720502     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (6)

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