Role of endotoxin in the hypermetabolic state after acute ethanol exposure

American Journal of Physiology - Gastrointestinal and Liver Physiology

Volumn 275, Issue 6 38-6, 1998, Pages

  Rivera, Chantal A ^a   Bradford, Blair U ^a   Seabra, Vitor ^a   Thurman, Ronald G ^a,b  

^a UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

^b Mary Ellen Jones Bldg CB# 7365 ^*  (United States)

Author keywords

Antibiotics; Eicosanoids; Gadolinium chloride; Kupffer cells; Liver

Indexed keywords

ALCOHOL; ANTIBIOTIC AGENT; ENDOTOXIN; GADOLINIUM CHLORIDE; ICOSANOID; PROSTAGLANDIN E2;

ALCOHOL METABOLISM; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENDOTOXEMIA; FEMALE; HYPERMETABOLISM; ICOSANOID METABOLISM; KUPFFER CELL; LIVER METABOLISM; NONHUMAN; OXYGEN CONSUMPTION; PRIORITY JOURNAL; RAT;

EID: 0032446710     PISSN: 01931857     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpgi.1998.275.6.g1252     Document Type: Article

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