Coronary artery disease is not associated with the E298→D variant of the constitutive, endothelial nitric oxide synthase gene [2]

Clinical Genetics

Volumn 54, Issue 6, 1998, Pages 528-529

  Liyou, N ^a   Simons, L ^a   Friedlander, Y ^a   Simons, J ^a   McCallum, J ^a   O'Shaughnessy, K ^a   Davis, D ^a   Johnson, A ^a  

^a UNIVERSITY OF QUEENSLAND   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

NITRIC OXIDE SYNTHASE;

AMINO ACID SUBSTITUTION; CONTROLLED STUDY; CORONARY ARTERY DISEASE; ENZYME POLYMORPHISM; HUMAN; LETTER; MAJOR CLINICAL STUDY; POINT MUTATION; PRIORITY JOURNAL; RESTRICTION FRAGMENT LENGTH POLYMORPHISM;

EID: 0032441578     PISSN: 00099163     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1399-0004.1998.tb03776.x     Document Type: Letter

References (8)

1. Complete amino acid sequence of the ADP/ATP carrier from beef heart mitochondri
2. Endothelium-derived nitric oxide in the control of tissue perfusion and oxygen supply: Physiological and pathophysiological implications
3. A common variant of the endothelial nitric oxide synthase gene is a risk factor for coronary atherosclerosis in the east Anglian region of the UK
4. Cloning and structural characterization of the human endothelial nitric-oxide-synthase gene
5. Study of the elderly: sociological and cardiovascular risk factors at entry
6. Risk factors for coronary heart disease in the prospective Dubbo Study of Australian elderly
7. Public health, cardiovascular disease and molecular biology
8. A smoking-dependent risk of coronary artery disease associated with a polymorphism of the endothelial nitric oxide synthase gene