Giant cell arteritis - A molecular approach to the multiple facets of the syndrome

Annales de Medecine Interne

Volumn 149, Issue 7, 1998, Pages 420-424

  Weyand, Cornelia M ^a  

^a MAYO CLINIC   (United States)

Author keywords

Cytokines; Pathogenesis; T cells; Vasculitis

Indexed keywords

GAMMA INTERFERON; INTERLEUKIN 2; PLATELET DERIVED GROWTH FACTOR;

ARTERY WALL; ARTICLE; CLINICAL FEATURE; GIANT CELL ARTERITIS; HUMAN; IMMUNOPATHOGENESIS; PATHOPHYSIOLOGY; RHEUMATIC POLYMYALGIA; SYNDROME DELINEATION; T LYMPHOCYTE;

BRAIN ISCHEMIA; CYTOKINES; GROWTH SUBSTANCES; HUMANS; LYMPHOCYTE ACTIVATION; MACROPHAGES; MOLECULAR BIOLOGY; PHENOTYPE; POLYMYALGIA RHEUMATICA; T-LYMPHOCYTES; TEMPORAL ARTERITIS;

EID: 0032438236     PISSN: 0003410X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (4)

1. HUNDER GG : Giant cell arteritis and polymyalgia rheumatica. Med Clin North Am, 1997 ; 81 : 195-219.
2. WEYAND CM, GORONZY JJ : Giant cell arteritis as an antigen driven disease. Rheum Dis Clin North Am, 1995 ; 21(4) : 1027-39.
3. BRACK A, GEISLER A, MARTINEZ-TABOADA V, YOUNGE B, GORONZY JJ, WEYAND CM : Giant cell vasculitis is a T cell dependent disease. Mol Med, 1997 ; 81 : 29-55.
4. WAGNER AD, BJORNSSON J, BARTLEY GB, GORONZY JJ, WEYAND CM : Interferon gamma producing T cells in giant cell vasculitis represent a minority of tissue infiltrating cells and are located distant from the site of pathology. Am J Pathol, 1996; 148: 1925-33.