|
|
Volumn 39, Issue 12, 1998, Pages 1383-
|
|
Hypothesis that tiagabine-induced NCSE is associated with GABAergic hyperfunction, with GABA(B) receptors playing a critical role, is supported by a case of generalized NCSE induced by baclofen.
a
a
NONE
|
|
Author keywords
[No Author keywords available]
|
|
Indexed keywords
4 AMINOBUTYRIC ACID RECEPTOR;
ANTICONVULSIVE AGENT;
BACLOFEN;
NIPECOTIC ACID DERIVATIVE;
TIAGABINE;
CHEMICALLY INDUCED DISORDER;
DRUG EFFECT;
EPILEPTIC STATE;
HUMAN;
NOTE;
PHYSIOLOGY;
ANTICONVULSANTS;
BACLOFEN;
HUMANS;
NIPECOTIC ACIDS;
RECEPTORS, GABA;
STATUS EPILEPTICUS;
MLCS;
MLOWN;
|
|
EID: 0032437144
PISSN: 00139580
EISSN: None
Source Type: Journal
DOI: 10.1111/j.1528-1157.1998.tb01345.x Document Type: Note |
|
Times cited : (7)
|
|
References (0)
|
