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Volumn 10, Issue 10, 1998, Pages 3201-3209
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Chronic hyperammonemia impairs the glutamate-nitric oxide-cyclic GMP pathway in cerebellar neurons in culture and in the rat in vivo
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Author keywords
Ammonia; Neurotoxicity; Nitric oxide synthase; NMDA receptor
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Indexed keywords
AMMONIA;
CALCIUM ION;
CYCLIC GMP;
GLUTAMIC ACID;
GUANYLATE CYCLASE;
N ACETYL S NITROSOPENICILLAMINE;
N METHYL DEXTRO ASPARTIC ACID;
N METHYL DEXTRO ASPARTIC ACID RECEPTOR;
NITRIC OXIDE SYNTHASE;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ARTICLE;
CALCIUM CELL LEVEL;
CELL DEATH;
CONTROLLED STUDY;
DOSE TIME EFFECT RELATION;
ENZYME ACTIVATION;
HEPATIC ENCEPHALOPATHY;
HYPERAMMONEMIA;
MALE;
MICRODIALYSIS;
NERVE CELL CULTURE;
NEUROTOXICITY;
NEWBORN;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
SIGNAL TRANSDUCTION;
AMMONIA;
ANIMALS;
CELL DEATH;
CELLS, CULTURED;
CEREBELLUM;
CYCLIC GMP;
DIALYSIS SOLUTIONS;
DOSE-RESPONSE RELATIONSHIP, DRUG;
EXTRACELLULAR SPACE;
GLUTAMIC ACID;
MALE;
MICRODIALYSIS;
N-METHYLASPARTATE;
NEURONS;
NITRIC OXIDE;
PERFUSION;
RATS;
RATS, WISTAR;
RECEPTORS, N-METHYL-D-ASPARTATE;
SIGNAL TRANSDUCTION;
TIME FACTORS;
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EID: 0032430914
PISSN: 0953816X
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1460-9568.1998.00329.x Document Type: Article |
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Times cited : (165)
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References (1)
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