-
1
-
-
0029935175
-
Selegiline
-
Anonymous
-
Anonymous, 1996. Selegiline. Drugs Fut. 21, 225.
-
(1996)
Drugs Fut.
, vol.21
, pp. 225
-
-
-
2
-
-
0028030607
-
Possible mechanisms of action of (-)deprenyl and other MAO-B inhibitors in some neurological and psychiatric disorders
-
Berry M.D., Juorio A.V., Paterson I.A. Possible mechanisms of action of (-)deprenyl and other MAO-B inhibitors in some neurological and psychiatric disorders. Prog. Neurobiol. 44:1994;141-161.
-
(1994)
Prog. Neurobiol.
, vol.44
, pp. 141-161
-
-
Berry, M.D.1
Juorio, A.V.2
Paterson, I.A.3
-
3
-
-
0027504033
-
Plasma 5-hydroxyindoleacetic acid as an indicator of monoamine oxidase-A inhibition in rat brain and peripheral tissues
-
Celada P., Artigas F. Plasma 5-hydroxyindoleacetic acid as an indicator of monoamine oxidase-A inhibition in rat brain and peripheral tissues. J. Neurochem. 61:1993;2191-2198.
-
(1993)
J. Neurochem.
, vol.61
, pp. 2191-2198
-
-
Celada, P.1
Artigas, F.2
-
4
-
-
0018777855
-
Does the B form selective monoamine oxidase inhibitor lose selectivity by long term treatment?
-
Ekstedt B., Magyar K., Knoll J. Does the B form selective monoamine oxidase inhibitor lose selectivity by long term treatment? Biochem. Pharmacol. 28:1978;919-923.
-
(1978)
Biochem. Pharmacol.
, vol.28
, pp. 919-923
-
-
Ekstedt, B.1
Magyar, K.2
Knoll, J.3
-
5
-
-
0028176919
-
Effect of L-deprenyl, its structural analogues and some monoamine oxidase inhibitors on dopamine uptake
-
Fang J., Yu P.H. Effect of L-deprenyl, its structural analogues and some monoamine oxidase inhibitors on dopamine uptake. Neuropharmacology. 33:1994;763-768.
-
(1994)
Neuropharmacology
, vol.33
, pp. 763-768
-
-
Fang, J.1
Yu, P.H.2
-
9
-
-
0030434805
-
L-Deprenyl (selegiline) decreases excitatory synaptic transmission in the rat hippocampus via a dopaminergic mechanism
-
Hsu K.S., Huang C.C., Su M.T., Tsai J.J. L-Deprenyl (selegiline) decreases excitatory synaptic transmission in the rat hippocampus via a dopaminergic mechanism. J. Pharmacol. Exp. Ther. 279:1996;740-747.
-
(1996)
J. Pharmacol. Exp. Ther.
, vol.279
, pp. 740-747
-
-
Hsu, K.S.1
Huang, C.C.2
Su, M.T.3
Tsai, J.J.4
-
10
-
-
0030979442
-
L-Deprenyl (selegiline) limits the repetitive firing of action potentials in rat hippocampal CA1 neurons via a dopaminergic mechanism
-
Huang C.C., Tsai J.J., Hsu K.S. L-Deprenyl (selegiline) limits the repetitive firing of action potentials in rat hippocampal CA1 neurons via a dopaminergic mechanism. Brain Res. 753:1997;27-35.
-
(1997)
Brain Res.
, vol.753
, pp. 27-35
-
-
Huang, C.C.1
Tsai, J.J.2
Hsu, K.S.3
-
11
-
-
0015694409
-
Central monoamines and convulsive thresholds in mice and rats
-
Kilian M., Frey H.-H. Central monoamines and convulsive thresholds in mice and rats. Neuropharmacology. 12:1973;681-692.
-
(1973)
Neuropharmacology
, vol.12
, pp. 681-692
-
-
Kilian, M.1
Frey, H.-H.2
-
12
-
-
0017874689
-
On the dual nature of monoamine oxidase
-
Knoll J. On the dual nature of monoamine oxidase. Horiz. Biochem. Biophys. 5:1978;37.
-
(1978)
Horiz. Biochem. Biophys.
, vol.5
, pp. 37
-
-
Knoll, J.1
-
13
-
-
0022964709
-
The pharmacology of (-)deprenyl
-
Knoll J. The pharmacology of (-)deprenyl. J. Neural Trans. 22(Suppl.):1986;75-89.
-
(1986)
J. Neural Trans.
, vol.22
, Issue.SUPPL.
, pp. 75-89
-
-
Knoll, J.1
-
14
-
-
0026520677
-
The pharmacological profile of (-)deprenyl (selegiline) and its relevance for humans - A personal view
-
Knoll J. The pharmacological profile of (-)deprenyl (selegiline) and its relevance for humans - a personal view. Pharmacol. Toxicol. 70:1992;317-321.
-
(1992)
Pharmacol. Toxicol.
, vol.70
, pp. 317-321
-
-
Knoll, J.1
-
15
-
-
0031907334
-
(-)Deprenyl (selegiline), a catecholaminergic activity enhancer (CAE) substance acting in the brain
-
Knoll J. (-)Deprenyl (selegiline), a catecholaminergic activity enhancer (CAE) substance acting in the brain. Pharmacol. Toxicol. 82:1998;57-66.
-
(1998)
Pharmacol. Toxicol.
, vol.82
, pp. 57-66
-
-
Knoll, J.1
-
16
-
-
0027940607
-
Multiple, small dose administration of (-)deprenyl enhances catecholaminergic activity and diminishes serotoninergic activity in the brain and these effects are unrelated to MAO-B inhibition
-
Knoll J., Miklya I. Multiple, small dose administration of (-)deprenyl enhances catecholaminergic activity and diminishes serotoninergic activity in the brain and these effects are unrelated to MAO-B inhibition. Arch. Int. Pharmacodyn. Ther. 328:1994;1-15.
-
(1994)
Arch. Int. Pharmacodyn. Ther.
, vol.328
, pp. 1-15
-
-
Knoll, J.1
Miklya, I.2
-
17
-
-
0029670154
-
(-)Deprenyl and (-)1-phenyl-2-propylaminopentane, ((-)PPAP) , act primarily as potent stimulants of action potential transmitter release coupling in the catecholaminergic neurons
-
Knoll J., Miklya I., Knoll B., Marko R., Kelemen K. (-)Deprenyl and (-)1-phenyl-2-propylaminopentane, ((-)PPAP) , act primarily as potent stimulants of action potential transmitter release coupling in the catecholaminergic neurons. Life Sci. 58:1996;817-827.
-
(1996)
Life Sci.
, vol.58
, pp. 817-827
-
-
Knoll, J.1
Miklya, I.2
Knoll, B.3
Marko, R.4
Kelemen, K.5
-
19
-
-
0345019797
-
New visions in the pharmacology of anticonvulsion
-
Löscher W. New visions in the pharmacology of anticonvulsion. Eur. J. Pharmacol. 342:1998;1-13.
-
(1998)
Eur. J. Pharmacol.
, vol.342
, pp. 1-13
-
-
Löscher, W.1
-
20
-
-
0029070492
-
Anticonvulsant and antiepileptogenic effect of L-deprenyl (selegiline) in the kindling model of epilepsy
-
Löscher W., Hönack D. Anticonvulsant and antiepileptogenic effect of L-deprenyl (selegiline) in the kindling model of epilepsy. J. Pharmacol. Exp. Ther. 274:1995;307-314.
-
(1995)
J. Pharmacol. Exp. Ther.
, vol.274
, pp. 307-314
-
-
Löscher, W.1
Hönack, D.2
-
21
-
-
0030432586
-
L-Deprenyl (selegiline) exerts anticonvulsant effects against different seizure types in mice
-
Löscher W., Lehmann H. L-Deprenyl (selegiline) exerts anticonvulsant effects against different seizure types in mice. J. Pharmacol. Exp. Ther. 277:1996;1410-1417.
-
(1996)
J. Pharmacol. Exp. Ther.
, vol.277
, pp. 1410-1417
-
-
Löscher, W.1
Lehmann, H.2
-
22
-
-
0023897574
-
Which animal models should be used in the search for new antiepileptic drugs? A proposal based on experimental and clinical considerations
-
Löscher W., Schmidt D. Which animal models should be used in the search for new antiepileptic drugs? A proposal based on experimental and clinical considerations. Epilepsy Res. 2:1988;145-181.
-
(1988)
Epilepsy Res.
, vol.2
, pp. 145-181
-
-
Löscher, W.1
Schmidt, D.2
-
23
-
-
0025817126
-
The role of technical, biological and pharmacological factors in the laboratory evaluation of anticonvulsant drug. III. Pentylenetetrazol seizure models
-
Löscher W., Hönack D., Fassbender C.P., Nolting B. The role of technical, biological and pharmacological factors in the laboratory evaluation of anticonvulsant drug. III. Pentylenetetrazol seizure models. Epilepsy Res. 8:1991;171-189.
-
(1991)
Epilepsy Res.
, vol.8
, pp. 171-189
-
-
Löscher, W.1
Hönack, D.2
Fassbender, C.P.3
Nolting, B.4
-
24
-
-
0002842768
-
Pharmacology of monoamine oxidase B inhibitors
-
In: Szelenyi, I. (Ed.) Birkhäuser, Basel
-
Magyar, K., 1993. Pharmacology of monoamine oxidase B inhibitors. In: Szelenyi, I. (Ed.), Inhibitors of Monoamine Oxidase B. Pharmacology and Clinical Use in Neurodegenerative Disorders. Birkhäuser, Basel, p. 125.
-
(1993)
Inhibitors of Monoamine Oxidase B. Pharmacology and Clinical Use in Neurodegenerative Disorders
, pp. 125
-
-
Magyar, K.1
-
26
-
-
0028803434
-
Pharmacokinetics and relative bioavailability of selegiline in healthy volunteers
-
Mahmood I., Marinac J.S., Willsie S., Mason W.D. Pharmacokinetics and relative bioavailability of selegiline in healthy volunteers. Biopharm. Drug Dispos. 16:1995;535-545.
-
(1995)
Biopharm. Drug Dispos.
, vol.16
, pp. 535-545
-
-
Mahmood, I.1
Marinac, J.S.2
Willsie, S.3
Mason, W.D.4
-
27
-
-
0030300166
-
Deprenyl in the treatment of Parkinson's disease: Clinical effects and speculations on mechanism of action
-
Olanow C.W. Deprenyl in the treatment of Parkinson's disease: clinical effects and speculations on mechanism of action. J. Neural Transm.-Suppl. 48:1996;75-84.
-
(1996)
J. Neural Transm.-Suppl.
, vol.48
, pp. 75-84
-
-
Olanow, C.W.1
-
28
-
-
0028182785
-
The effect of 6-months L-deprenyl administration on pineal MAO-A and MAO-B activity and on the content of melatonin and related indoles in aged female Fisher 344N rats
-
Oxenkrug G.F., Requintina P.J., Correa R.M., Yuwiler A. The effect of 6-months L-deprenyl administration on pineal MAO-A and MAO-B activity and on the content of melatonin and related indoles in aged female Fisher 344N rats. J. Neural Trans. 41(Suppl.):1994;249-252.
-
(1994)
J. Neural Trans.
, vol.41
, Issue.SUPPL.
, pp. 249-252
-
-
Oxenkrug, G.F.1
Requintina, P.J.2
Correa, R.M.3
Yuwiler, A.4
-
29
-
-
0001430156
-
Monoamines and the pathophysiology of seizure disorders
-
In: Frey, H.-H., Janz, D. (Eds.) Springer, Berlin
-
Przegalinski, E., 1985. Monoamines and the pathophysiology of seizure disorders. In: Frey, H.-H., Janz, D. (Eds.), Antiepileptic Drugs. Springer, Berlin, p. 101.
-
(1985)
Antiepileptic Drugs
, pp. 101
-
-
Przegalinski, E.1
-
30
-
-
0028590052
-
Therapy with L-deprenyl (selegiline) and relation to abuse liability
-
Schneider L.S., Tariot P.N., Goldstein B. Therapy with L-deprenyl (selegiline) and relation to abuse liability. Clin. Pharmacol. Ther. 56:1994;750-756.
-
(1994)
Clin. Pharmacol. Ther.
, vol.56
, pp. 750-756
-
-
Schneider, L.S.1
Tariot, P.N.2
Goldstein, B.3
-
31
-
-
0029731378
-
Modulation of gene expression rather than monoamine oxidase inhibition: (-)-deprenyl-related compounds in controlling neurodegeneration
-
Tatton W.G., Chalmersredman R.M.E. Modulation of gene expression rather than monoamine oxidase inhibition: (-)-deprenyl-related compounds in controlling neurodegeneration. Neurology. 47:1996;S171-S183.
-
(1996)
Neurology
, vol.47
-
-
Tatton, W.G.1
Chalmersredman, R.M.E.2
-
32
-
-
0030300164
-
(-)-Deprenyl reduces neuronal apoptosis and facilitates neuronal outgrowth by altering protein synthesis without inhibiting monoamine oxidase
-
Tatton W.G., Wadia J.S., Ju W.Y.H., Chalmersredman R.M.E., Tatton N.A. (-)-Deprenyl reduces neuronal apoptosis and facilitates neuronal outgrowth by altering protein synthesis without inhibiting monoamine oxidase. J. Neural Transm.-Suppl. 48:1996;45-59.
-
(1996)
J. Neural Transm.-Suppl.
, vol.48
, pp. 45-59
-
-
Tatton, W.G.1
Wadia, J.S.2
Ju, W.Y.H.3
Chalmersredman, R.M.E.4
Tatton, N.A.5
|