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Volumn 12, Issue 6, 1998, Pages 815-824

Stimulation of Jun N-terminal kinase (JNK) by gonadotropin-releasing hormone in pituitary αt3-1 cell line is mediated by protein kinase C, c-Src, and CDC42

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM ION; CELL CYCLE PROTEIN; G PROTEIN COUPLED RECEPTOR; GONADORELIN; LIGAND; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHOLIPASE; PROTEIN G; PROTEIN KINASE C; STRESS ACTIVATED PROTEIN KINASE; TUMOR PROMOTER;

EID: 0032230247     PISSN: 08888809     EISSN: None     Source Type: Journal    
DOI: 10.1210/mend.12.6.0120     Document Type: Article
Times cited : (120)

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    • The sites of phosphorylation by protein kinase C and an intact SH2 domain are required for the enhanced response to beta-adrenergic agonists in cells overexpressing c-src
    • (1993) Mol Cell Biol , vol.13 , pp. 2391-2400
    • Moyers, J.S.1    Bouton, A.H.2    Parsons, S.J.3
  • 46
    • 0030926774 scopus 로고    scopus 로고
    • Focal adhesion kinase overexpression enhances ras-dependent integrin signaling to ERK2/mitogen-activated protein kinase through interactions with and activation of c-Src
    • (1997) J Biol Chem , vol.272 , pp. 13189-13195
    • Schlaepfer, D.D.1    Hunter, T.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.