Specific targeting of ganglion cell sprouts provides an additional mechanism for restoring peripheral motor circuits in pelvic ganglia after spinal nerve damage

Journal of Neuroscience

Volumn 18, Issue 19, 1998, Pages 7987-7995

  Kepper, Mark E ^a   Keast, Janet R ^a  

^a UNIVERSITY OF QUEENSLAND   (Australia)

Author keywords

Autonomic ganglion; Deafferentation; Nerve injury; Parasympathetic; Pelvic reflexes; Plasticity; Sympathetic

Indexed keywords

ARTICLE; AUTONOMIC GANGLION; CELL COMMUNICATION AND CELL CONTACT; NERVE CELL GROWTH; NERVE FIBER GROWTH; NERVE INJURY; PLASTICITY; PRIORITY JOURNAL; SPINAL NERVE;

EID: 0032190175     PISSN: 02706474     EISSN: None     Source Type: Journal    
DOI: 10.1523/jneurosci.18-19-07987.1998     Document Type: Article

References (5)

1. Baron R, Jänig W (1988) Neurons projecting rostrally in the hypogastric nerve of the cat. J Auton Nerv Syst 24:81-86.
2. Causing CG, Gloster A, Aloyz R, Bamji SX, Chang E, Fawcett J, Kuchel G, Miller FD (1997) Synaptic innervation density is regulated by neuron-derived BDNF. Neuron 18:257-267.
3. Dail WG, Evan AP (1978) Effects of chronic deafferentation on adrenergic ganglion cells and small intensely fluorescent cells. J Neurocytol 7:25-37.
4. Dail WG, Minorsky N (1986) Composition of the pelvic nerve. Exp Neurol 92:278-283.
5. Dail WG, Moll MA, Weber K (1983) Localization of vasoactive intestinal polypeptide in penile erectile tissue and in the major pelvic ganglion of the rat. Neuroscience 10:1379-1386.