Systemic inflammation in patients with heart failure

European Heart Journal

Volumn 19, Issue 5, 1998, Pages 761-765

  Hasper, D ^a   Hummel, M ^a   Kleber, F X ^a   Reindl, I ^a   Volk, Hans Dieter ^a,b  

^a HUMBOLDT UNIVERSITY   (Germany)

^b CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

Author keywords

Adhesion molecules; Biventricular assist device system; Cardiogenic shock; Chronic heart failure; Cytokines; Systemic inflammatory response syndrome

Indexed keywords

CYTOKINE; ENDOTHELIAL LEUKOCYTE ADHESION MOLECULE 1; INTERLEUKIN 6; INTERLEUKIN 8; L SELECTIN; PADGEM PROTEIN; TUMOR NECROSIS FACTOR ALPHA; VASCULAR CELL ADHESION MOLECULE 1;

ADOLESCENT; ADULT; ARTICLE; ASSISTED CIRCULATION; BLOOD LEVEL; CARDIOGENIC SHOCK; CLINICAL ARTICLE; CONTROLLED STUDY; CORONARY ARTERY DISEASE; HEART FAILURE; HUMAN; HYPOXIA; INFECTION; INFLAMMATION; PATIENT MONITORING; PRIORITY JOURNAL;

ADULT; AGED; CELL ADHESION MOLECULES; CHRONIC DISEASE; CYTOKINES; FEMALE; HEART FAILURE, CONGESTIVE; HEART-ASSIST DEVICES; HUMANS; MALE; MIDDLE AGED; PROGNOSIS; SYSTEMIC INFLAMMATORY RESPONSE SYNDROME;

EID: 0032080919     PISSN: 0195668X     EISSN: None     Source Type: Journal    
DOI: 10.1053/euhj.1997.0858     Document Type: Article

References (17)

1. [1] Shreeniwas R, Koga S, Karakurum M et al. Hypoxia mediated induction of endothelial cell interleukin-1 alpha. An autocrine mechanism promoting expression of leukocyte adhesion molecules on the vessel surface. J Clin Invest 1992; 90: 2333-9.
2. [2] Scannell G, Waxman K, Kaml GJ et al. Hypoxia induces a human macrophage cell line to release tumor necrosis factor-alpha and its soluble receptors in vitro. J Surg Res 1993; 54: 218-25.
3. [3] Ertel W, Singh G, Morrison MH, Ayala A, Chaudry IH. Chemically induced hypotension increases PGE2 release and depresses macrophage antigen presentation. Am J Physiol 1993; 264: R655-60.
4. [4] Wang P, Ba ZF, Chaudry IH. Endothelial dysfunction occurs early following trauma-haemorrhage and persists despite fluid resuscitation. Am J Physiol 1993; 265: H973-9.
5. [5] Murray KD, Myerowitz PD, Watson KM et al. Effect of a total artificial heart on adaptive hormonal responses in humans with end-stage heart failure. Trans Am Soc Artif Intern Organs 1989; 35: 229-31.
6. [6] Bittner HB, Diemel KD, Friedel N, Stengele B, Hetzer R. Skin microcirculation and laser Doppler blood flow in patients suported by the Berlin heart biventricular assist device. Vasa 1992; 21: 149-53.
7. [7] Wiedermann CJ, Beimpold H, Herold M, Knapp E, Braunsteiner H. Increased levels of serum neopterin and decreased production of neutrophil superoxid anions in chronic heart failure with elevated levels of tumor necrosis factor-alpha. J Am Coll Cardiol 1993; 22: 1897-901.
8. [8] Dutka DP, Elborn JS, Delamere F, Shale DJ, Morris GK. Tumor necrosis factor alpha in severe congestive heart failure. Br Heart J 1993; 70: 141-3.
9. [9] Ferrari R, Bachetti T, Confortini R et al. Tumor necrosis factor soluble receptors in patients with varying degrees of congestive heart failure. Circulation 1995; 92: 1479-86.
10. [10] Munger MA, Johnson B, Amber IJ, Callahan KS, Gilbert EM. Circulating concentrations of proinflammatory cytokines in mild or moderate heart failure secondary to ischemic or idiopathic dilated cardiomyopathie. Am J Cardiol 1996; 77: 723-7.
11. [11] Torre-Amione G, Kapadia S, Lee J et al. Tumor necrosis factor-alpha and tumor necrosis factor receptors in the failing heart. Circulation 1996; 93: 704-11.
12. [12] Torre-Amione G, Kapadia S, Benedict C, Oral H, Young JB, Mann DL. Profinflammatory cytokine levels in patients with depressed left ventricular ejection fraction: a report from the Studies of Left Ventricular Dysfunction (SOLVD). J Am Coll Cardiol 1996; 27: 1201-6.
13. [13] Benjamin E, Leibowitz AB, Oropello J et al. Systemic hypoxic and inflammatory syndrome: An alternative designation for 'sepsis syndrome'. Crit Care Med 1992; 20: 680-3.
14. [14] Chaudry IH, Ayala A, Meldrum D et al. Hemorrhage induced alterations in cell mediated immune function. In: Faist E, Meakins J, Schildberg FW, eds. Host Defence Dysfunction in Trauma, Shock and Sepsis. Berlin-Heidelberg: Springer, 1993: 149-59.
15. [15] Kinugawa K, Takahashi T, Kohmoto O et al. Nitric oxide-mediated effects of interleukin-6 on [Ca2+]i and cell contraction in cultured chick ventricular myocytes. Circ Res 1994; 75: 285-95.
16. [16] Ungureanu-Longrois D, Balligand JL, Kelly RA, Smith TW. Myocardial contractile dysfunction in the systemic inflammatory response syndrome: role of cytokine inducible nitric oxide synthase in cardiac myocytes. J Mol Cell Cardiol 1995; 27: 155-67.
17. [17] Chaudry IH, Ayala A. Mechanism of increased susceptibility to infection following hemorrhage. Am J Surg 1993; 165: 59-67.