Kainic acid causes redox changes in cerebral cortex extracellular fluid: NMDA receptor activity increases ascorbic acid whereas seizure activity increases uric acid

Neuropharmacology

Volumn 37, Issue 2, 1998, Pages 149-157

  Layton, Matthew E ^a   Samson, Fred E ^b   Pazdernik, Thomas L ^b,c  

^a UNIVERSITY OF WASHINGTON MEDICAL CENTER   (United States)

^b UNIVERSITY OF KANSAS MEDICAL CENTER   (United States)

^c UNIVERSITY OF KANSAS SCHOOL OF MEDICINE   (United States)

Author keywords

Antioxidants; APV; Ascorbic acid; Kainic acid; Microdialysis; Midazolam; NMDA; Seizures; Uric acid

Indexed keywords

2 AMINO 5 PHOSPHONOVALERIC ACID; ASCORBIC ACID; KAINIC ACID; MIDAZOLAM; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; URIC ACID;

ANIMAL EXPERIMENT; ARTICLE; BRAIN CORTEX; CONCENTRATION RESPONSE; CONTROLLED STUDY; DRUG EFFECT; EXTRACELLULAR FLUID; HIGH PERFORMANCE LIQUID CHROMATOGRAPHY; MALE; MICRODIALYSIS; NONHUMAN; OXIDATION REDUCTION STATE; PRIORITY JOURNAL; RAT; SEIZURE;

ANIMALS; ANTIOXIDANTS; ASCORBIC ACID; CEREBRAL CORTEX; EXCITATORY AMINO ACID AGONISTS; EXTRACELLULAR SPACE; KAINIC ACID; MALE; MICRODIALYSIS; MIDAZOLAM; OXIDATION-REDUCTION; RATS; RATS, WISTAR; RECEPTORS, N-METHYL-D-ASPARTATE; SEIZURES; URIC ACID;

EID: 0032004201     PISSN: 00283908     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0028-3908(98)00002-1     Document Type: Article

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