Anoxia during kainate status epilepticus shortens behavioral convulsions but generates hippocampal neuron loss and supragranular mossy fiber sprouting

Epilepsy Research

Volumn 30, Issue 2, 1998, Pages 133-151

  Mathern, Gary W ^a,b   Price, Greg ^a   Rosales, Carlos ^a,b   Pretorius, James K ^b   Lozada, Alana ^b   Mendoza, Delia ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Epilepsy; Excitotoxicity; NIH image; Synaptic reorganization

Indexed keywords

ANIMAL EXPERIMENT; ANIMAL TISSUE; ANOXIA; ARTICLE; BRAIN BLOOD FLOW; BRAIN OXYGEN CONSUMPTION; CONTROLLED STUDY; EPILEPTIC STATE; HIPPOCAMPUS; MALE; MOSSY FIBER; NERVE FIBER GROWTH; NONHUMAN; PRIORITY JOURNAL; RAT;

ANIMALS; BEHAVIOR, ANIMAL; CELL COUNT; EXCITATORY AMINO ACID AGONISTS; HIPPOCAMPUS; HISTOCYTOCHEMISTRY; HYPOXIA, BRAIN; KAINIC ACID; MALE; MOSSY FIBERS, HIPPOCAMPAL; NEURONS; RATS; RATS, SPRAGUE-DAWLEY; SEIZURES; STATUS EPILEPTICUS;

EID: 0031971459     PISSN: 09201211     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0920-1211(97)00103-4     Document Type: Article

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