The role of glutamatergic neurotransmission in the pathophysiology of alcoholism

Annual Review of Medicine

Volumn 49, Issue , 1998, Pages 173-184

  Tsai, Guochuan ^a   Coyle, Joseph T ^a  

^a MASSACHUSETTS GENERAL HOSPITAL   (United States)

Author keywords

Ethanol; Excitotoxicity; Glutamate; N methyl D aspartate

Indexed keywords

ALCOHOL; GLUTAMIC ACID; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; NEUROTRANSMITTER;

ALCOHOL ABUSE; ALCOHOLISM; BEHAVIOR MODIFICATION; EXCITABILITY; FETAL ALCOHOL SYNDROME; HUMAN; NERVE CELL NECROSIS; PATHOPHYSIOLOGY; PRIORITY JOURNAL; REVIEW;

ALCOHOL WITHDRAWAL DELIRIUM; ALCOHOLISM; BRAIN; CELL DEATH; CENTRAL NERVOUS SYSTEM DEPRESSANTS; ETHANOL; EXCITATORY AMINO ACID ANTAGONISTS; FETAL ALCOHOL SYNDROME; GLUTAMIC ACID; HUMANS; NEURONS; NEUROTOXINS; NEUROTRANSMITTER AGENTS; RECEPTORS, N-METHYL-D-ASPARTATE; SEIZURES; SYNAPTIC TRANSMISSION; UP-REGULATION;

EID: 0031959272     PISSN: 00664219     EISSN: None     Source Type: Book Series    
DOI: 10.1146/annurev.med.49.1.173     Document Type: Review

References (2)

1. Nevo I, Hamon M. 1995. Neurotransmitter and neuromodulatory mechanisms involved in alcohol abuse and alcoholism. Neurochem. Int. 26:305-36
2. Nestoros JN. 1980. Ethanol specifically potentiates GABA-mediated neurotransmission in feline cerebral cortex. Science 209:708-10