Interactions between Epstein-Barr virus and the cell cycle control machinery

Histology and Histopathology

Volumn 13, Issue 2, 1998, Pages 461-467

  Sinclair, A J ^a  

^a UNIVERSITY OF SUSSEX   (United Kingdom)

Author keywords

Cancer; Carcinoma; Cell cycle; Epstein Bart Virus; Leukaemia; Lymphoma

Indexed keywords

BURKITT LYMPHOMA; CELL CYCLE; EPSTEIN BARR VIRUS; HUMAN; LEUKEMIA; NASOPHARYNX CARCINOMA; NONHUMAN; REVIEW;

CELL CYCLE; HERPESVIRUS 4, HUMAN; HUMANS; PHENOTYPE;

HUMAN HERPESVIRUS 4;

EID: 0031958884     PISSN: 02133911     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Review

References (9)

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3. Arvanitakis L., Yaseen N. and Sharma S. (1995). Latent membrane protein-1 induces cyclin D2 expression, pRb hyperphosphorylation, and loss of TGF-beta-1-mediated growth- inhibition in EBV-positive B-cells. J. Immunol. 155, 1047-1056.
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6. Cannell E.J., Farrell P.J. and Sinclair A.J. (1996). Epstein-Barr-virus exploits the normal-cell pathway to regulate Rb activity during the immortalization of primary B-cells. Oncogene 13, 1413-1421.
7. Cayrol C. and Remington E. (1996a). G(0)/G(1), growth arrest mediated by a region encompassing the basic leucine-zipper (bzip) domain of the Epstein-Barr-virus transactivator Zta. J. Biol. Chem. 271, 31799-31802.
8. Cayrol C. and Flemington E. K. (1996b). The Epstein-Barr-virus bzip transcription factor Zta causes G(0)/G(1) cell-cycle arrest through induction of cyclin-dependent kinase inhibitors. EMBO J. 15, 2748-2759.
9. Zhang Z., Gutsch D. and Kenney, S. (1994). Functional and physical interaction between p53 and BZLF1: implications for Epstein-Barr virus latency. J. Virol. 14, 1929-1938.