Programming of the hypothalamo-pituitary-adrenal axis and the fetal origins of adult disease hypothesis

European Journal of Pediatrics, Supplement

Volumn 157, Issue 1, 1998, Pages

  Clark, P M ^a  

^a UNIVERSITY OF SOUTHAMPTON   (United Kingdom)

Author keywords

Blood pressure; Fetal growth; Glucose tolerance; HPA axis; Programming

Indexed keywords

DEXAMETHASONE; GLUCOCORTICOID;

BLOOD PRESSURE; CONFERENCE PAPER; FETUS DISEASE; FETUS GROWTH; GLUCOSE INTOLERANCE; HUMAN; HYDROCORTISONE BLOOD LEVEL; HYPOPHYSIS ADRENAL SYSTEM; PATHOGENESIS; PATHOPHYSIOLOGY; PRENATAL EXPOSURE; PRIORITY JOURNAL;

ANIMALS; BIRTH WEIGHT; BLOOD PRESSURE; CHILD; EMBRYONIC AND FETAL DEVELOPMENT; FEMALE; GLUCOCORTICOIDS; GLUCOSE INTOLERANCE; HUMANS; HYDROXYSTEROID DEHYDROGENASES; HYPOTHALAMO-HYPOPHYSEAL SYSTEM; INFANT, LOW BIRTH WEIGHT; INFANT, NEWBORN; INFANT, PREMATURE; MATERNAL-FETAL EXCHANGE; PITUITARY-ADRENAL SYSTEM; PLACENTA; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; RATS;

EID: 0031932259     PISSN: 09439676     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Conference Paper

References (2)

1. Barker DJP, Osmond C (1986) Infant mortality, childhood nutrition, and ischaemic heart disease in England and Wales. Lancet 1:1077-1081
2. Benediktsson R, Lindsay RS, Noble J, Seckl JR, Edwards CRW (1993) Glucocorticoid exposure in utero: new model for adult hypertension. Lancet 341:339-341