Hypoxia enhances proliferation and generation of IP3 in pulmonary artery fibroblasts but not in those from the mesenteric circulation

Chest

Volumn 114, Issue 1 SUPPL., 1998, Pages

  Peacock, Andrew J ^b   Scott, P ^a,b   Plevin, R ^a,b   Wadsworth, R ^a,b   Welsh, D ^a,b  

^a WESTERN INFIRMARY   (United Kingdom)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

INOSITOL TRISPHOSPHATE;

ANIMAL CELL; CATTLE; CELL PROLIFERATION; CONFERENCE PAPER; GROWTH STIMULATION; HYPOXIA; LUNG ARTERY; LUNG FIBROBLAST; MESENTERY BLOOD FLOW; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PULMONARY HYPERTENSION; SIGNAL TRANSDUCTION; VASODILATATION;

EID: 0031829349     PISSN: 00123692     EISSN: None     Source Type: Journal    
DOI: 10.1378/chest.114.1_supplement.24s     Document Type: Conference Paper

References (2)

1. 3 generation and DNA synthesis in bovine pulmonary artery fibroblasts. Am J Respir Crit Care Med 1996; 153:A576
2. Palmer S, Hughes KT, Lee DY, et al. Development of a novel, Ins(1,4,5)P3-specific binding assay: its use to determine the intracellular concentration of Ins(1,4,5)P3 in unstimulated and vasopressin-stimulated rat hepatocytes. Cell Signal 1989; 1:147-56