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Volumn 18, Issue 4, 1998, Pages 117-122
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Pharmacological profiles of absence seizure-induced increases in CRE- and AP-1 DNA-binding activities in γ-butyrolactone-treated mice
a
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Author keywords
Absence seizure; DNA binding; GABA(B) receptors; GBL; Thalamus
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Indexed keywords
3 AMINOPROPYL(CYCLOHEXYLMETHYL)PHOSPHINIC ACID;
3 AMINOPROPYL(DIETHOXYMETHYL)PHOSPHINIC ACID;
3 AMINOPROPYL(HEXYL)PHOSPHINIC ACID;
4 AMINOBUTYRIC ACID B RECEPTOR;
4 AMINOBUTYRIC ACID B RECEPTOR BLOCKING AGENT;
CGP 46381;
ETHOSUXIMIDE;
GAMMA BUTYROLACTONE;
TRANSCRIPTION FACTOR AP 1;
UNCLASSIFIED DRUG;
ABSENCE;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
AUTOMATISM;
BRAIN CORTEX;
CONTROLLED STUDY;
CYCLIC AMP RESPONSIVE ELEMENT;
GABAERGIC SYSTEM;
INTRAPERITONEAL DRUG ADMINISTRATION;
MALE;
MOUSE;
NONHUMAN;
PROTEIN DNA BINDING;
THALAMOCORTICAL TRACT;
THALAMUS;
4-BUTYROLACTONE;
ACTIVATING TRANSCRIPTION FACTOR 2;
ANIMALS;
ANTICONVULSANTS;
BRAIN;
BRAIN CHEMISTRY;
CEREBRAL CORTEX;
CYCLIC AMP RESPONSE ELEMENT-BINDING PROTEIN;
DISEASE MODELS, ANIMAL;
DNA-BINDING PROTEINS;
EPILEPSY, ABSENCE;
ETHOSUXIMIDE;
GABA ANTAGONISTS;
MICE;
ORGANOPHOSPHORUS COMPOUNDS;
PHOSPHINIC ACIDS;
THALAMUS;
TRANSCRIPTION FACTOR AP-1;
TRANSCRIPTION FACTORS;
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EID: 0031766387
PISSN: 13402544
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
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Times cited : (5)
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References (32)
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