C4AQ0 superimposed on a primary defect increases the susceptibility to systemic lupus erythematosus (SLE) in a family with association between C4AQ0 and SLE

Journal of Rheumatology

Volumn 25, Issue 11, 1998, Pages 2118-2125

  Traustadóttir, Kristín H ^a   Steinsson, Kristján ^b,c   Erlendsson, Kristján ^a,d  

^a Department of Immunology ^*

^b Division of Rheumatology

^c Center for Rheumatology Research

^d LANDSPITALI UNIVERSITY HOSPITAL   (Iceland)

Author keywords

C4aq0; Families; Pathogenesis; Systemic lupus erythematosus

Indexed keywords

COMPLEMENT COMPONENT C3D; COMPLEMENT COMPONENT C4A;

ANTIGEN ANTIBODY COMPLEX; ARTICLE; CLINICAL ARTICLE; COMPARATIVE STUDY; COMPLEMENT DEFICIENCY; COMPLEMENT SYSTEM; DISEASE ASSOCIATION; DISEASE PREDISPOSITION; ERYTHROCYTE; FAMILIAL INCIDENCE; GENE DELETION; GENE MUTATION; HUMAN; OPSONIZATION; PATHOGENESIS; PRIORITY JOURNAL; SEROLOGY; SYSTEMIC LUPUS ERYTHEMATOSUS;

ANTIGEN-ANTIBODY COMPLEX; COMPLEMENT C4A; ERYTHROCYTES; FEMALE; GENETIC PREDISPOSITION TO DISEASE; HETEROZYGOTE; HOMOZYGOTE; HUMANS; LUPUS ERYTHEMATOSUS, SYSTEMIC; MALE; PEDIGREE; REFERENCE VALUES;

EID: 0031763561     PISSN: 0315162X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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