Crouzon-like craniofacial dysmorphology in the mouse is caused by an insertional mutation at the Fgf3/Fgf4 locus

Developmental Dynamics

Volumn 212, Issue 2, 1998, Pages 242-249

  Carlton, Mark B L ^a   Colledge, William H ^b   Evans, Martin J ^a  

^a UNIVERSITY OF CAMBRIDGE   (United Kingdom)

^b UNIVERSITY OF CAMBRIDGE   (United Kingdom)

Author keywords

Craniosynostosis; Murine; Retroviral insertion mutagenesis

Indexed keywords

FIBROBLAST GROWTH FACTOR; FIBROBLAST GROWTH FACTOR RECEPTOR; VIRUS VECTOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; AUTOSOMAL DOMINANT DISORDER; CONTROLLED STUDY; CRANIOFACIAL MALFORMATION; CRANIOFACIAL SYNOSTOSIS; CROUZON SYNDROME; FEMALE; GENE EXPRESSION REGULATION; GENE INSERTION; GENE LOCUS; GENE MUTATION; MALE; MOUSE; NONHUMAN; PEDIGREE; PRIORITY JOURNAL; PROTEIN DOMAIN; RETROVIRUS; SIGNAL TRANSDUCTION;

ANIMALS; BASE SEQUENCE; CRANIOFACIAL DYSOSTOSIS; DISEASE MODELS, ANIMAL; DNA PRIMERS; FEMALE; FIBROBLAST GROWTH FACTOR 3; FIBROBLAST GROWTH FACTOR 4; FIBROBLAST GROWTH FACTORS; GENETIC TECHNIQUES; HETEROZYGOTE; HUMANS; MALE; MICE; MUTAGENESIS, INSERTIONAL; MUTATION; PHENOTYPE; POLYMERASE CHAIN REACTION; PROTO-ONCOGENE PROTEINS;

ANIMALIA; MAMMALIA; MURINAE; RODENTIA; UNIDENTIFIED RETROVIRUS;

EID: 0031749743     PISSN: 10588388     EISSN: None     Source Type: Journal    
DOI: 10.1002/(SICI)1097-0177(199806)212:2<242::AID-AJA8>3.0.CO;2-H     Document Type: Article

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