On a mechanism for isochromatid 17q in a subset of Ph+ chronic myeloid leukemia patients

Medical Hypotheses

Volumn 50, Issue 5, 1998, Pages 375-383

  Matioli, G T ^a  

^a UNIVERSITY OF SOUTHERN CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ARTICLE; CANCER GENETICS; CHROMOSOME 17Q; CHRONIC MYELOID LEUKEMIA; HUMAN; ISOCHROMOSOME; PHILADELPHIA 1 CHROMOSOME; PRIORITY JOURNAL; TUMOR SUPPRESSOR GENE;

EID: 0031747848     PISSN: 03069877     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0306-9877(98)90208-0     Document Type: Article

References (9)

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3. Matioli G T. On the translation of a soliton-like kink along the stack of a coiled chromatid. Med Hypotheses 1993; 41: 256.
4. Mitelman F. Catalogue of chromosomal aberrations in cancer. Cytog Cell Genet 1983; 36: 5.
5. Mitelman F. Catalogue of Chromosomal Aberrations in Cancer. New York: Wiley-Liss, 1994.
6. Matioli G T. Variational (Lagrangian) approach to myeloid differentiation path(s). Med Hypothese 1995; 47: 327.
7. Borgstrom G H, Vuopio P, de la Chapelle A. Abnormalities of chromosome No. 17 in myeloproliferative disorders. Cancer Genet Cytogenet 1982; 5: 123.
8. Matioli G T. On monosegmental sister chromatid exchanges (S.C.E.) Med Hypotheses 1991; 36: 318.
9. Fugazza G, Bruzzone R, Puppo L, Sessarego M. Granulocytes with segmented nucleus retain normal chromosome 17 in Philadelphia chromosome-positive chronic myeloid leukemia with i(17q) and pseudo-Pelger anomaly. Cancer Genet Cytogenet 1996; 90: 166.