Short note: The fetal origins hypothesis: Linking pineal gland hypoplasia with coronary heart disease and stroke

Medical Hypotheses

Volumn 50, Issue 4, 1998, Pages 357-358

  Maurizi, C P ^a  

^a Houston Medical Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MELATONIN;

ANTIOXIDANT ACTIVITY; CEREBROVASCULAR ACCIDENT; CORONARY ARTERY DISEASE; FETUS; HUMAN; HYPOPLASIA; INFANT; ISCHEMIC HEART DISEASE; MALNUTRITION; NEWBORN; PINEAL BODY; PRIORITY JOURNAL; SHORT SURVEY; STROKE; SUDDEN INFANT DEATH SYNDROME;

EID: 0031744819     PISSN: 03069877     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0306-9877(98)90012-3     Document Type: Article

References (12)

1. Barker D J P. Fetal origins of coronary heart disease. Br Med J 1995; 311: 171-174.
2. Kennaway D J, Goble F C, Stamp G E. Factors influencing the development of melatonin rhythmicity in humans. J Clin Endocrinol Metab 1996; 81: 1525-1532.
3. Maurizi C P. Could exogenous melatonin prevent sudden infant death syndrome? Med Hypotheses 1997; 49: 425-427.
4. Stein C E, Fall C H D, Kumaran K, Osmond C, Cox V, Barker D J P. Fetal growth and coronary heart disease in South India. Lancet 1996; 348: 1269-1273.
5. Martyn C N, Barker D J P, Osmond C. Mothers' pelvic size, fetal growth, and death from stroke and coronary heart disease in men in the UK. Lancet 1996; 348: 1264-1268.
6. Brugger P, Markti W, Herold M. Impaired nocturnal secretion of melatonin in coronary heart disease. Lancet 1995; 345: 1408.
7. Fiorina P, Lattuada G, Ponari O, Silvestrini C, Dall'Aglio P. Impaired nocturnal melatonin excretion and changes of immunological status in ischaemic stroke patients. Lancet 1996; 347: 692-693.
8. Hoffman R M, Garewal H S. Antioxidants and the prevention of coronary heart disease. Arch Intern Med 1995; 155: 241-246.
9. Witztum J L. The oxidation hypothesis of atherosclerosis. Lancet 1994; 344: 793-795.
10. Bjorkerud S, Bjorkerud B. Apoptosis is abundant in human atherosclerotic lesions, especially in inflammatory cells (macrophages and T cells), and may contribute to the accumulation of gruel and plaque instability. Am J Pathol 1996; 149: 367-380.
11. Lizard G, Deckert V, Dubrez L, Moisant M, Gambert P. Lagrost. Induction of apoptosis in endothelial cells treated with cholesterol oxides. Am J Pathol 1996; 148: 1625-1638.
12. Reiter R J, Pablos M I, Agapito T T, Guerrero J M. Melatonin in the context of the free radical theory of aging. Ann NY Acad Sci 1996; 786: 362-378.