-
1
-
-
0004275062
-
-
of special interest. W.C. Parks, Mecham R.P. Academic Press San Diego Comprehensive and up-to-date reviews on many aspects of MMP biology and chemistry
-
of special interest Parks WC, Mecham RP. Matrix metalloproteinases. 1998;Academic Press, San Diego, Comprehensive and up-to-date reviews on many aspects of MMP biology and chemistry.
-
(1998)
Matrix Metalloproteinases
-
-
-
2
-
-
0030825453
-
Matrix metalloproteinase inhibitors in the treatment of cancer
-
Brown PD. Matrix metalloproteinase inhibitors in the treatment of cancer. Med Oncol. 14:1997;1-10.
-
(1997)
Med Oncol
, vol.14
, pp. 1-10
-
-
Brown, P.D.1
-
3
-
-
0028945864
-
A miracle enough: The power of mice
-
Piagen K. A miracle enough: the power of mice. Nat Med. 1:1995;215-220.
-
(1995)
Nat Med
, vol.1
, pp. 215-220
-
-
Piagen, K.1
-
4
-
-
0030890685
-
Mighty mice: Transgenic technology "knocks out" questions of matrix metalloproteinase function
-
Shapiro SD. Mighty mice: transgenic technology "knocks out" questions of matrix metalloproteinase function. Matrix Biol. 15:1997;527-533.
-
(1997)
Matrix Biol
, vol.15
, pp. 527-533
-
-
Shapiro, S.D.1
-
5
-
-
0029993449
-
Know your neighbors: Three phenotypes in null mutants of the myogenic bHLH gene MRF4
-
Olson EN, Arnold H-H, Rigby PWJ, Wold BJ. Know your neighbors: three phenotypes in null mutants of the myogenic bHLH gene MRF4. Cell. 85:1996;1-4.
-
(1996)
Cell
, vol.85
, pp. 1-4
-
-
Olson, E.N.1
Arnold H-H2
Rigby, P.W.J.3
Wold, B.J.4
-
7
-
-
0346963598
-
MMP-9/gelatinase B is a key regulator of growth plate angiogenesis and apoptosis of hypertrophic chondrocytes
-
of special interest. This initial description of MMP-9-deficient mice demonstrates the the role of MMP-9 in long bone development. MMP-9 induces primary angiogenesis associated with vascular invasion of the cartilage matrix that precedes endochondral ossification
-
Vu TH, Shipley JM, Bergers G, Berger JE, Helms JA, Hanahan D, Shapiro SD, Senior RM, Werb Z. MMP-9/gelatinase B is a key regulator of growth plate angiogenesis and apoptosis of hypertrophic chondrocytes. of special interest Cell. 93:1998;411-422 This initial description of MMP-9-deficient mice demonstrates the the role of MMP-9 in long bone development. MMP-9 induces primary angiogenesis associated with vascular invasion of the cartilage matrix that precedes endochondral ossification.
-
(1998)
Cell
, vol.93
, pp. 411-422
-
-
Vu, T.H.1
Shipley, J.M.2
Bergers, G.3
Berger, J.E.4
Helms, J.A.5
Hanahan, D.6
Shapiro, S.D.7
Senior, R.M.8
Werb, Z.9
-
8
-
-
0030764771
-
Matrix metalloproteinases MMP2 and MMP9 are produced in early stages of kidney morphogenesis but only MMP9 is required for renal organogenesis in vitro
-
Lelongt B, Trugnan G, Murphy G, Ronco PM. Matrix metalloproteinases MMP2 and MMP9 are produced in early stages of kidney morphogenesis but only MMP9 is required for renal organogenesis in vitro. J Cell Biol. 136:1997;1363-1373.
-
(1997)
J Cell Biol
, vol.136
, pp. 1363-1373
-
-
Lelongt, B.1
Trugnan, G.2
Murphy, G.3
Ronco, P.M.4
-
10
-
-
0029054734
-
Molecular bases of the acute coronary syndromes
-
Libby P. Molecular bases of the acute coronary syndromes. Circulation. 91:1995;2844-2850.
-
(1995)
Circulation
, vol.91
, pp. 2844-2850
-
-
Libby, P.1
-
11
-
-
0029115532
-
Production and localization of 92-kD gelatinase in abdominal aortic aneurysms: An elastolytic metalloproteinase expressed by aneurysm-infiltrating macrophages
-
Thompson RW, Mertens RA, Liao S, Holmes DR, Mecham RP, Welgus HG, Parks WC. Production and localization of 92-kD gelatinase in abdominal aortic aneurysms: an elastolytic metalloproteinase expressed by aneurysm-infiltrating macrophages. J Clin Invest. 96:1995;318-326.
-
(1995)
J Clin Invest
, vol.96
, pp. 318-326
-
-
Thompson, R.W.1
Mertens, R.A.2
Liao, S.3
Holmes, D.R.4
Mecham, R.P.5
Welgus, H.G.6
Parks, W.C.7
-
12
-
-
0030024169
-
Mouse models of atherosclerosis
-
Breslow J. Mouse models of atherosclerosis. Science. 272:1996;685-688.
-
(1996)
Science
, vol.272
, pp. 685-688
-
-
Breslow, J.1
-
13
-
-
16944363984
-
Plasmin predisposes to atherosclerotic aneurysm formation by activation of matrix metalloproteinases
-
-/- mice also lacking urokinase-type (but not tissue-type) plasminogen activators are protected from macrophage penetration of elastic lamina and microaneurysm formation. This paper contains the most direct evidence to date that plasmin generated by urokinase-type plasminogen activators is required for pro-MMP activation in vivo
-
-/- mice also lacking urokinase-type (but not tissue-type) plasminogen activators are protected from macrophage penetration of elastic lamina and microaneurysm formation. This paper contains the most direct evidence to date that plasmin generated by urokinase-type plasminogen activators is required for pro-MMP activation in vivo.
-
(1997)
Nat Genet
, vol.17
, pp. 439-444
-
-
Carmeleit, P.1
Moons, L.2
Lijnen, R.3
Crawley, J.4
Tipping, P.5
Drew, A.6
Eeckhout, Y.7
Shapiro, S.D.8
Lupu, F.9
Collen, D.10
-
14
-
-
0029911694
-
Metalloelastase is required for macrophage-mediated proteolysis and matrix invasion in mice
-
Shipley JM, Wesselschmidt RL, Kobayshi DK, Ley TJ, Shapiro SD. Metalloelastase is required for macrophage-mediated proteolysis and matrix invasion in mice. Proc Natl Acad Sci USA. 93:1996;3942-3946.
-
(1996)
Proc Natl Acad Sci USA
, vol.93
, pp. 3942-3946
-
-
Shipley, J.M.1
Wesselschmidt, R.L.2
Kobayshi, D.K.3
Ley, T.J.4
Shapiro, S.D.5
-
15
-
-
15844406034
-
Progression of coronary atherosclerosis is associated with a common genetic variant of the human stromelysin-1 promoter which results in reduced gene expression
-
Ye S, Eriksson P, Hamsten A, Kurkinen M, Humphries SE, Henney AM. Progression of coronary atherosclerosis is associated with a common genetic variant of the human stromelysin-1 promoter which results in reduced gene expression. J Biol Chem. 271:1996;13055-13060.
-
(1996)
J Biol Chem
, vol.271
, pp. 13055-13060
-
-
Ye, S.1
Eriksson, P.2
Hamsten, A.3
Kurkinen, M.4
Humphries, S.E.5
Henney, A.M.6
-
16
-
-
0030293598
-
Matrix metalloproteinases and the development of cancer
-
Coussens LM, Werb Z. Matrix metalloproteinases and the development of cancer. Chem Biol. 3:1996;895-904.
-
(1996)
Chem Biol
, vol.3
, pp. 895-904
-
-
Coussens, L.M.1
Werb, Z.2
-
17
-
-
0029991531
-
Complex roles of matrix metalloproteinases in tumor progression
-
Powell WC, Matrisian LM. Complex roles of matrix metalloproteinases in tumor progression. Curr Top Microbiol Immunol. 213:1996;1-21.
-
(1996)
Curr Top Microbiol Immunol
, vol.213
, pp. 1-21
-
-
Powell, W.C.1
Matrisian, L.M.2
-
18
-
-
0031036521
-
Stimulation of matrix metalloproteinase production by recombinant extracellular matrix metalloproteinase inducer from transfected Chinese hamster ovary cells
-
of special interest. Previously, this group found that extracellular matrix metalloproteinase inducer (EMMPRIN), a transmembrane glycoprotein attached to the surface of many types of malignant human tumor cells, stimulated matrix metalloproteinase (MMP) synthesis in fibroblasts. This paper, continuing Biswas' pioneering studies post-humously, shows that CHO cells transfected with EMMPRIN post-translationally process the molecule and this stimulates human fibroblast production of MMPs-1, -2, and -3
-
Guo H, Zucker S, Gordon MK, Toole BP, Biswas C. Stimulation of matrix metalloproteinase production by recombinant extracellular matrix metalloproteinase inducer from transfected Chinese hamster ovary cells. of special interest J Biol Chem. 272:1997;24-27 Previously, this group found that extracellular matrix metalloproteinase inducer (EMMPRIN), a transmembrane glycoprotein attached to the surface of many types of malignant human tumor cells, stimulated matrix metalloproteinase (MMP) synthesis in fibroblasts. This paper, continuing Biswas' pioneering studies post-humously, shows that CHO cells transfected with EMMPRIN post-translationally process the molecule and this stimulates human fibroblast production of MMPs-1, -2, and -3.
-
(1997)
J Biol Chem
, vol.272
, pp. 24-27
-
-
Guo, H.1
Zucker, S.2
Gordon, M.K.3
Toole, B.P.4
Biswas, C.5
-
19
-
-
15844420283
-
Localization of matrix metalloproteinase MMP-2 to the surface of invasive cells by interaction with integrin αvβ3
-
Brooks PC, Stromblad S, Sanders LC, von Schalscha TL, Aimes RT, Stetler-Stevenson WG, Quigley JP, Cheresh DA. Localization of matrix metalloproteinase MMP-2 to the surface of invasive cells by interaction with integrin αVβ3. Cell. 85:1996;683-693.
-
(1996)
Cell
, vol.85
, pp. 683-693
-
-
Brooks, P.C.1
Stromblad, S.2
Sanders, L.C.3
Von Schalscha, T.L.4
Aimes, R.T.5
Stetler-Stevenson, W.G.6
Quigley, J.P.7
Cheresh, D.A.8
-
20
-
-
0010712792
-
Disruption of angiogenesis by PEX, a noncatalytic metalloproteinase fragment with integrin binding activity
-
of special interest. Data in [17] demonstrated that MMP-2 binds to tumor cell αVβ3 integrin and confirmed the relationship of this event to tumor progression. This report takes us further, showing that dissociation of the carboxy-terminal domain from the catalytic domain occurs in vivo and the free carboxyl terminus both activates pro-MMP-2 and competes with MMP-2 tumor cell binding, inhibiting growth
-
Brooks PC, Silletti S, von Schalscha TL, Friedlander M, Cheresh DA. Disruption of angiogenesis by PEX, a noncatalytic metalloproteinase fragment with integrin binding activity. of special interest Cell. 92:1998;391-400 Data in [17] demonstrated that MMP-2 binds to tumor cell αVβ3 integrin and confirmed the relationship of this event to tumor progression. This report takes us further, showing that dissociation of the carboxy-terminal domain from the catalytic domain occurs in vivo and the free carboxyl terminus both activates pro-MMP-2 and competes with MMP-2 tumor cell binding, inhibiting growth.
-
(1998)
Cell
, vol.92
, pp. 391-400
-
-
Brooks, P.C.1
Silletti, S.2
Von Schalscha, T.L.3
Friedlander, M.4
Cheresh, D.A.5
-
21
-
-
0032031861
-
Reduced angiogenesis and tumor progression in gelatinase A-deficient mice
-
Itoh T, Tanioka M, Yoshida H, Yoshioka T, Nishimoto H, Itohara S. Reduced angiogenesis and tumor progression in gelatinase A-deficient mice. Cancer Res. 58:1998;1048-1051.
-
(1998)
Cancer Res
, vol.58
, pp. 1048-1051
-
-
Itoh, T.1
Tanioka, M.2
Yoshida, H.3
Yoshioka, T.4
Nishimoto, H.5
Itohara, S.6
-
22
-
-
0031036359
-
Intestinal tumorigenesis is suppressed in mice lacking the metalloproteinase matrilysin
-
of special interest. This report and [21], utilizing gene-targeted mice, provide evidence for involvement of matrix metalloproteinases directly in the process of carcinogenesis, rather than merely 'clearing the way' for tumors to move
-
Wilson CL, Heppner KJ, Labosky PA, Hogan BL, Matrisian LM. Intestinal tumorigenesis is suppressed in mice lacking the metalloproteinase matrilysin. of special interest Proc Natl Acad Sci USA. 94:1997;1402-1407 This report and [21], utilizing gene-targeted mice, provide evidence for involvement of matrix metalloproteinases directly in the process of carcinogenesis, rather than merely 'clearing the way' for tumors to move.
-
(1997)
Proc Natl Acad Sci USA
, vol.94
, pp. 1402-1407
-
-
Wilson, C.L.1
Heppner, K.J.2
Labosky, P.A.3
Hogan, B.L.4
Matrisian, L.M.5
-
23
-
-
0032559821
-
In vivo evidence that the stromelysin-3 metalloproteinase contributes in a paracrine manner to epithelial cell malignancy
-
of special interest. See annotation to [20]
-
Masson R, Lefebvre O, Noel A, El Fahime M, Chenared M-P, Wendling C, Kebers F, LeMeur M, Dierich A, Foidart J-M, et al. In vivo evidence that the stromelysin-3 metalloproteinase contributes in a paracrine manner to epithelial cell malignancy. of special interest J Cell Biol. 140:1998;1535-1541 See annotation to [20].
-
(1998)
J Cell Biol
, vol.140
, pp. 1535-1541
-
-
Masson, R.1
Lefebvre, O.2
Noel, A.3
El Fahime, M.4
Chenared M-P5
Wendling, C.6
Kebers, F.7
Lemeur, M.8
Dierich, A.9
Foidart J-M10
-
24
-
-
0029836160
-
Inhibition of SV40 T antigen-induced hepatocellular carcinoma in TIMP-1 transgenic mice
-
Martin DC, Ruther U, Sanchez-Sweatman OH, Orr FW, Khokha R. Inhibition of SV40 T antigen-induced hepatocellular carcinoma in TIMP-1 transgenic mice. Oncogene. 13:1996;569-576.
-
(1996)
Oncogene
, vol.13
, pp. 569-576
-
-
Martin, D.C.1
Ruther, U.2
Sanchez-Sweatman, O.H.3
Orr, F.W.4
Khokha, R.5
-
25
-
-
0030012534
-
Overexpression of tissue inhibitor of metalloproteinases-2 retroviral-mediated gene transfer in vivo inhibits tumor growth and invasion
-
Imren S, Kohn DB, Shimada H, Blavier L, DeClerck YA. Overexpression of tissue inhibitor of metalloproteinases-2 retroviral-mediated gene transfer in vivo inhibits tumor growth and invasion. Cancer Res. 56:1996;2891-2895.
-
(1996)
Cancer Res
, vol.56
, pp. 2891-2895
-
-
Imren, S.1
Kohn, D.B.2
Shimada, H.3
Blavier, L.4
Declerck, Y.A.5
-
26
-
-
0030471877
-
Targeted mutagenesis of Timp-1 reveals that lung tumor invasion is influenced by Timp-1 genotype of the tumor but not by that of the host
-
Soloway PD, Alexander CM, Werb Z, Jaenisch R. Targeted mutagenesis of Timp-1 reveals that lung tumor invasion is influenced by Timp-1 genotype of the tumor but not by that of the host. Oncogene. 13:1996;2307-2314.
-
(1996)
Oncogene
, vol.13
, pp. 2307-2314
-
-
Soloway, P.D.1
Alexander, C.M.2
Werb, Z.3
Jaenisch, R.4
-
27
-
-
0027970092
-
A novel angiogenesis inhibitor which mediates the suppression of metastasis by a Lewis lung carcinoma
-
O'Reilly MS, Holmgren L, Shing Y, Chen C, Rosenthal RA, Moses M, Lane WS, Cao Y, Sage EH, Folkman J. A novel angiogenesis inhibitor which mediates the suppression of metastasis by a Lewis lung carcinoma. Cell. 79:1994;315-328.
-
(1994)
Cell
, vol.79
, pp. 315-328
-
-
O'Reilly, M.S.1
Holmgren, L.2
Shing, Y.3
Chen, C.4
Rosenthal, R.A.5
Moses, M.6
Lane, W.S.7
Cao, Y.8
Sage, E.H.9
Folkman, J.10
-
28
-
-
0030998660
-
Macrophage-derived metalloelastase is responsible for the generation of angiostatin in Lewis lung carcinoma
-
of special interest. This report provided the first evidence for proteolytic generation of angiostatin from plasminogen. Expression of macrophage elastase correlated with angiostatin generation in Lewis lung carcinoma primary tumors
-
Dong Z, Kumar R, Yang X, Fidler IJ. Macrophage-derived metalloelastase is responsible for the generation of angiostatin in Lewis lung carcinoma. of special interest Cell. 88:1997;801-810 This report provided the first evidence for proteolytic generation of angiostatin from plasminogen. Expression of macrophage elastase correlated with angiostatin generation in Lewis lung carcinoma primary tumors.
-
(1997)
Cell
, vol.88
, pp. 801-810
-
-
Dong, Z.1
Kumar, R.2
Yang, X.3
Fidler, I.J.4
-
29
-
-
15444352707
-
Angiostatin-converting enzyme activities of human matrilysin (MMP-7) and gelatinase B/type IV collagenase (MMP-9)
-
Patterson BC, Sang QA. Angiostatin-converting enzyme activities of human matrilysin (MMP-7) and gelatinase B/type IV collagenase (MMP-9). J Biol Chem. 272:1997;28823-28825.
-
(1997)
J Biol Chem
, vol.272
, pp. 28823-28825
-
-
Patterson, B.C.1
Sang, Q.A.2
-
30
-
-
0345578183
-
Generation of angiostatin by matrix metalloproteinases: Effects on neovascularization
-
in press
-
Cornelius LA, Nehring L, Klein B, Pierce R, Bolinski M, Welgus HG, Shapiro SD. Generation of angiostatin by matrix metalloproteinases: effects on neovascularization. J Immunol. 1998;. in press.
-
(1998)
J Immunol
-
-
Cornelius, L.A.1
Nehring, L.2
Klein, B.3
Pierce, R.4
Bolinski, M.5
Welgus, H.G.6
Shapiro, S.D.7
-
31
-
-
0030930671
-
Kringle 5 of plasminogen is a novel inhibitor of endothelial cell growth
-
Cao Y, Chen A, An SSA, Ji RW, Davidson D, Llinas M. Kringle 5 of plasminogen is a novel inhibitor of endothelial cell growth. J Biol Chem. 272:1997;22924-22928.
-
(1997)
J Biol Chem
, vol.272
, pp. 22924-22928
-
-
Cao, Y.1
Chen, A.2
An, S.S.A.3
Ji, R.W.4
Davidson, D.5
Llinas, M.6
-
32
-
-
0030861045
-
Generation of angiostatin by reduction and proteolysis of plasmin: Catalysis by a plasmin reductase secreted by cultured cells
-
of special interest. This paper not only demonstrates the capacity of reduced plasmin to generate angiostatin from its parent plasminogen, but also gives the first example of an extracellular reduction of a protein disulfide bond
-
Stathakis P, Fitzgerald M, Matthias LJ, Chesterman CN, Hogg PJ. Generation of angiostatin by reduction and proteolysis of plasmin: catalysis by a plasmin reductase secreted by cultured cells. of special interest J Biol Chem. 272:1997;20641-20645 This paper not only demonstrates the capacity of reduced plasmin to generate angiostatin from its parent plasminogen, but also gives the first example of an extracellular reduction of a protein disulfide bond.
-
(1997)
J Biol Chem
, vol.272
, pp. 20641-20645
-
-
Stathakis, P.1
Fitzgerald, M.2
Matthias, L.J.3
Chesterman, C.N.4
Hogg, P.J.5
-
33
-
-
10344247677
-
Human prostate carcinoma cells express enzymatic activity that converts human plasminogen to the angiogenesis inhibitor, angiostatin
-
Gately S, Twardowski SP, Stack MS, Patrick M, Boggio L, Cundiff DL, Schnaper HW, Madison L, Volpert O, Bouck N, et al. Human prostate carcinoma cells express enzymatic activity that converts human plasminogen to the angiogenesis inhibitor, angiostatin. Cancer Res. 56:1996;4887-4890.
-
(1996)
Cancer Res
, vol.56
, pp. 4887-4890
-
-
Gately, S.1
Twardowski, S.P.2
Stack, M.S.3
Patrick, M.4
Boggio, L.5
Cundiff, D.L.6
Schnaper, H.W.7
Madison, L.8
Volpert, O.9
Bouck, N.10
-
34
-
-
0031454617
-
Endostatin: An endogenous inhibitor of angiogenesis and tumor growth
-
of special interest. Continuing the theme that protein fragments inhibit angiogenesis, endostatin, a 20 kDa carboxy-terminal fragment of collagen XVIII, was identified from hemangioendotheliomas. Endostatin inhibited endothelial cell proliferation in vitro and angiogenesis and tumor growth in vivo
-
O'Reilly M, Boehm T, Shing Y, Fukai N, Vasios G, Lane WS, Flynn E, Birkhead JR, Olsen BR, Folkman J. Endostatin: An endogenous inhibitor of angiogenesis and tumor growth. of special interest Cell. 88:1997;277-285 Continuing the theme that protein fragments inhibit angiogenesis, endostatin, a 20 kDa carboxy-terminal fragment of collagen XVIII, was identified from hemangioendotheliomas. Endostatin inhibited endothelial cell proliferation in vitro and angiogenesis and tumor growth in vivo.
-
(1997)
Cell
, vol.88
, pp. 277-285
-
-
O'Reilly, M.1
Boehm, T.2
Shing, Y.3
Fukai, N.4
Vasios, G.5
Lane, W.S.6
Flynn, E.7
Birkhead, J.R.8
Olsen, B.R.9
Folkman, J.10
-
35
-
-
0031469819
-
Antiangiogenic therapy of experimental cancer does not induce acquired drug resistance
-
of special interest. Treatment of Lewis lung cell carcinomas, T241 fibrosarcomas, and B16 melanomas in mice with endostatin resulted in prolonged tumor dormancy. No drug resistance occurred and there was no tumor recurrence after several cycles of repeated therapy
-
Boehm T, Folkman J, Browder T, O'Reilly MS. Antiangiogenic therapy of experimental cancer does not induce acquired drug resistance. of special interest Nature. 390:1997;404-407 Treatment of Lewis lung cell carcinomas, T241 fibrosarcomas, and B16 melanomas in mice with endostatin resulted in prolonged tumor dormancy. No drug resistance occurred and there was no tumor recurrence after several cycles of repeated therapy.
-
(1997)
Nature
, vol.390
, pp. 404-407
-
-
Boehm, T.1
Folkman, J.2
Browder, T.3
O'Reilly, M.S.4
-
36
-
-
0030042145
-
Combination therapy including a gelatinase inhibitor and cytotoxic agent reduces local invasion and metastasis of murine Lewis lung carcinoma
-
Anderson IC, Shipp MA, Docherty AJP, Teicher BA. Combination therapy including a gelatinase inhibitor and cytotoxic agent reduces local invasion and metastasis of murine Lewis lung carcinoma. Cancer Res. 56:1996;710-715.
-
(1996)
Cancer Res
, vol.56
, pp. 710-715
-
-
Anderson, I.C.1
Shipp, M.A.2
Docherty, A.J.P.3
Teicher, B.A.4
-
37
-
-
0030823772
-
Macrophage elastase is required for cigarette smoke-induced emphysema in mice
-
-/- mice provide a model for macrophage proteolysis, demonstrating a critical role of macrophages in emphysema and unmasking a proteinase-dependent mechanism of inflammatory cell recruitment
-
-/- mice provide a model for macrophage proteolysis, demonstrating a critical role of macrophages in emphysema and unmasking a proteinase-dependent mechanism of inflammatory cell recruitment.
-
(1997)
Science
, vol.277
, pp. 2002-2004
-
-
Hautamaki, R.D.1
Kobayashi, D.K.2
Senior, R.M.3
Shapiro, S.D.4
-
39
-
-
0019519757
-
Elastin fragments attract macrophage precursors to diseased sites in pulmonary emphysema
-
Hunninghake GW, Davidson JM, Rennard S, Szapiel S, Gadek JE, Crystal RG. Elastin fragments attract macrophage precursors to diseased sites in pulmonary emphysema. Science. 212:1981;925-927.
-
(1981)
Science
, vol.212
, pp. 925-927
-
-
Hunninghake, G.W.1
Davidson, J.M.2
Rennard, S.3
Szapiel, S.4
Gadek, J.E.5
Crystal, R.G.6
-
40
-
-
0021170197
-
Val-Gly-Val-Ala-Pro-Gly, a repeating peptide in elastin, is chemotactic for fibroblasts and monocytes
-
Senior RM, Griffin GL, Mecham RP, Wrenn DS, Prasad KU, Urry DW. Val-Gly-Val-Ala-Pro-Gly, a repeating peptide in elastin, is chemotactic for fibroblasts and monocytes. J Cell Biol. 99:1984;870-874.
-
(1984)
J Cell Biol
, vol.99
, pp. 870-874
-
-
Senior, R.M.1
Griffin, G.L.2
Mecham, R.P.3
Wrenn, D.S.4
Prasad, K.U.5
Urry, D.W.6
-
41
-
-
0032479967
-
Gelatinase B-deficient mice are resistant to experimental bullous pemphigold
-
in press
-
Liu Z, Shipley JM, Vu T, Zhou X, Diaz LA, Werb Z, Senior RM. Gelatinase B-deficient mice are resistant to experimental bullous pemphigold. J Exp Med. 1998;. in press.
-
(1998)
J Exp Med
-
-
Liu, Z.1
Shipley, J.M.2
Vu, T.3
Zhou, X.4
Diaz, L.A.5
Werb, Z.6
Senior, R.M.7
-
42
-
-
0028302990
-
Targeted expression of stromelysin-1 in mammary gland provides for a role of proteinases in branching morphogenesis and the requirement for an intact basement membrane for tissue-specific gene expression
-
Sympson CJ, Talhouk RS, Alexander CM, Chin JR, Clift SM, Bissell MJ. Targeted expression of stromelysin-1 in mammary gland provides for a role of proteinases in branching morphogenesis and the requirement for an intact basement membrane for tissue-specific gene expression. J Cell Biol. 125:1994;681-693.
-
(1994)
J Cell Biol
, vol.125
, pp. 681-693
-
-
Sympson, C.J.1
Talhouk, R.S.2
Alexander, C.M.3
Chin, J.R.4
Clift, S.M.5
Bissell, M.J.6
-
43
-
-
0030798679
-
Induction of cell migration by matrix metalloprotease-2 cleavage of laminin-5
-
of special interest. This article demonstrates the existence of pro-motility cryptic sites of laminin specific for gelatinase A. This study and [1] suggest that local proteinase concentration and matrix degradation determine cell behavior
-
Giannelli G, Falk-Marzillier J, Schiraldi O, Stetler-Stevenson WG, Quaranta V. Induction of cell migration by matrix metalloprotease-2 cleavage of laminin-5. of special interest Science. 277:1997;225-228 This article demonstrates the existence of pro-motility cryptic sites of laminin specific for gelatinase A. This study and [1] suggest that local proteinase concentration and matrix degradation determine cell behavior.
-
(1997)
Science
, vol.277
, pp. 225-228
-
-
Giannelli, G.1
Falk-Marzillier, J.2
Schiraldi, O.3
Stetler-Stevenson, W.G.4
Quaranta, V.5
-
44
-
-
0030761314
-
Induction and repression of collagenase-1 by keratinocytes is controlled by distinct components of different extracellular matrix compartments
-
of special interest. See annotation to [45]
-
Sudbeck BD, Pilcher BK, Welgus HG, Parks WC. Induction and repression of collagenase-1 by keratinocytes is controlled by distinct components of different extracellular matrix compartments. of special interest J Biol Chem. 272:1997;22103-22110 See annotation to [45].
-
(1997)
J Biol Chem
, vol.272
, pp. 22103-22110
-
-
Sudbeck, B.D.1
Pilcher, B.K.2
Welgus, H.G.3
Parks, W.C.4
-
45
-
-
0030949650
-
The activity of collagenase-1 is required for keratinocyte migration on a type I collagen matrix
-
of special interest. This reference and [44] build a story of how keratinocyte interaction with collagen in a provisional wound matrix initiate re-epithelialization
-
Pilcher BK, Dumin JA, Sudbeck BD, Krane SM, Welgus HG, Parks WC. The activity of collagenase-1 is required for keratinocyte migration on a type I collagen matrix. of special interest J Cell Biol. 137:1997;1445-1457 This reference and [44] build a story of how keratinocyte interaction with collagen in a provisional wound matrix initiate re-epithelialization.
-
(1997)
J Cell Biol
, vol.137
, pp. 1445-1457
-
-
Pilcher, B.K.1
Dumin, J.A.2
Sudbeck, B.D.3
Krane, S.M.4
Welgus, H.G.5
Parks, W.C.6
-
46
-
-
0029079272
-
Components of the nuclear signaling cascade that regulate collagenase gene expression in response to integrin-derived signals
-
Tremble P, Damsky CH, Werb Z. Components of the nuclear signaling cascade that regulate collagenase gene expression in response to integrin-derived signals. J Cell Biol. 129:1995;1707-1720.
-
(1995)
J Cell Biol
, vol.129
, pp. 1707-1720
-
-
Tremble, P.1
Damsky, C.H.2
Werb, Z.3
-
47
-
-
0030715322
-
ECM and cell surface proteolysis: Regulating cellular ecology
-
of special interest. This is an excellent review highlighting how proteolysis of matrix at the cell surface influences a variety of cell functions
-
Werb Z. ECM and cell surface proteolysis: Regulating cellular ecology. of special interest Cell. 91:1997;439-442 This is an excellent review highlighting how proteolysis of matrix at the cell surface influences a variety of cell functions.
-
(1997)
Cell
, vol.91
, pp. 439-442
-
-
Werb, Z.1
-
48
-
-
0030908028
-
Latent transforming growth factor-beta: Structural features and mechanisms of activation
-
Munger JS, Harpel JG, Gleizes PE, Mazzieri R, Nunes I, Rifkin DB. Latent transforming growth factor-beta: structural features and mechanisms of activation. Kidney Int. 51:1997;1376-1382.
-
(1997)
Kidney Int
, vol.51
, pp. 1376-1382
-
-
Munger, J.S.1
Harpel, J.G.2
Gleizes, P.E.3
Mazzieri, R.4
Nunes, I.5
Rifkin, D.B.6
-
49
-
-
0030884231
-
Unaltered secretion of β-amyloid precursor protein in gelatinase A (MMP-2)-deficient mice
-
Itoh T, Ikeda T, Gomi H, Nakao S, Suzuki T, Itohara S. Unaltered secretion of β-amyloid precursor protein in gelatinase A (MMP-2)-deficient mice. J Biol Chem. 272:1997;22389-22392.
-
(1997)
J Biol Chem
, vol.272
, pp. 22389-22392
-
-
Itoh, T.1
Ikeda, T.2
Gomi, H.3
Nakao, S.4
Suzuki, T.5
Itohara, S.6
-
50
-
-
14444285039
-
Susceptibility of stromelysin 1-deficient mice to collagen-induced arthritis and cartilage destruction
-
Mudgett JS, Hutchinson NI, Chartrain NA, Forsyth AJ, McDonnell J, Singer II, Bayne EK, Flanagan J, Kawka D, Shen CF, et al. Susceptibility of stromelysin 1-deficient mice to collagen-induced arthritis and cartilage destruction. Arthritis Rheum. 41:1998;110-121.
-
(1998)
Arthritis Rheum
, vol.41
, pp. 110-121
-
-
Mudgett, J.S.1
Hutchinson, N.I.2
Chartrain, N.A.3
Forsyth, A.J.4
McDonnell, J.5
Singer, I.I.6
Bayne, E.K.7
Flanagan, J.8
Kawka, D.9
Shen, C.F.10
-
51
-
-
0029002420
-
A targeted mutation at the known collagenase cleavage site in mouse type I collagen impairs tissue remodeling
-
Liu X, Wu H, Byrne M, Jeffrey J, Krane S, Jaenisch R. A targeted mutation at the known collagenase cleavage site in mouse type I collagen impairs tissue remodeling. J Cell Biol. 130:1995;227-237.
-
(1995)
J Cell Biol
, vol.130
, pp. 227-237
-
-
Liu, X.1
Wu, H.2
Byrne, M.3
Jeffrey, J.4
Krane, S.5
Jaenisch, R.6
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