Retroviral vector-mediated transfer of an antisense cyclin G1 construct inhibits osteosarcoma tumor growth in nude mice

Human Gene Therapy

Volumn 8, Issue 14, 1997, Pages 1667-1674

  Chen, Daniel S ^a  

^a UNIVERSITY OF SOUTHERN CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CANCER INHIBITION; CELL CYCLE G1 PHASE; CELL CYCLE G2 PHASE; CELL CYCLE S PHASE; CONTROLLED STUDY; GENE EXPRESSION REGULATION; GENE THERAPY; GENE TRANSFER; HUMAN; HUMAN CELL; METASTASIS; MITOSIS; NONHUMAN; NUDE MOUSE; OSTEOSARCOMA; RETROVIRUS; SUBCUTANEOUS TISSUE TUMOR; SUPERNATANT; TISSUE SECTION;

EID: 0031580665     PISSN: 10430342     EISSN: None     Source Type: Journal    
DOI: 10.1089/hum.1997.8.14-1667     Document Type: Article

References (4)

1. DAHLIN, D., and COVENTRY, M. (1967). Osteogenic sarcoma: A study of six hundred cases. J. Bone Joint Surg. 49, 101-110.
2. DAMRON, T.A., and PRITCHARD, D.J. (1995). Current combined treatment of high-grade osteosarcomas. Oncology 9, 327-340.
3. ELLEDGE, S.J., and HARPER, J.W. (1994). Cdk inhibitors: On the threshold of checkpoints and development. Curr. Opin. Cell Biol. 6, 874-878.
4. HALL, F.L., and VULLIET, P.R. (1991). Proline-directed protein phosphorylation and cell cycle regulation. Curr. Opin. Cell Biol. 3, 176-184.