In CA1 hippocampal neurons, the redox state of the NMDARs determines LTP expressed by NMDA but not by AMPARs
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Long-term potentiation in the hippocampal CA1 region in the presence of N-methyl-D-aspartate receptor antagonists
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2+ accumulations and metabotropic glutamate receptor activation associated with an N-methyl-D-aspartate receptor-independent long-term potentiation in hippocampal CA1 neurons
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