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Volumn 29, Issue 4, 1997, Pages 375-382
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Host mechanisms: Are they the key to the various clinical outcomes of Helicobacter pylori infection?
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Author keywords
Duodenal ulcer; Gastric acid; Helicobacter pylori; Stomach neoplasm
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Indexed keywords
ANTIOXIDANT;
CYTOKINE;
HISTAMINE;
INTERLEUKIN 1BETA;
SOMATOSTATIN;
TUMOR NECROSIS FACTOR ALPHA;
ALCOHOL CONSUMPTION;
ATROPHY;
BACTERIAL VIRULENCE;
DIET;
DISEASE PREDISPOSITION;
DUODENUM PH;
ENDOCRINE CELL;
ENTEROCHROMAFFIN CELL;
EXOCRINE CELL;
GASTRIN CELL;
GASTRITIS;
GENETICS;
GRAM NEGATIVE INFECTION;
HEALTH STATUS;
HELICOBACTER PYLORI;
HOST RESISTANCE;
HUMAN;
IMMUNE RESPONSE;
NONHUMAN;
REVIEW;
SALT INTAKE;
SMOKING;
STOMACH ACID SECRETION;
STOMACH ANTRUM;
STOMACH CANCER;
STOMACH PARIETAL CELL;
CYTOKINES;
DUODENAL DISEASES;
GASTRIC ACID;
GASTRIN-SECRETING CELLS;
HELICOBACTER INFECTIONS;
HELICOBACTER PYLORI;
HISTAMINE;
HUMANS;
HYDROGEN-ION CONCENTRATION;
PARIETAL CELLS, GASTRIC;
SOMATOSTATIN-SECRETING CELLS;
STOMACH DISEASES;
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EID: 0031461026
PISSN: 11258055
EISSN: None
Source Type: Journal
DOI: None Document Type: Review |
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Times cited : (7)
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References (91)
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