Pinacidil's effects on defibrillation outcomes: Role of increased potassium conductance via the K(ATP) channel

Journal of Cardiovascular Pharmacology and Therapeutics

Volumn 2, Issue 3, 1997, Pages 171-180

  Winecoff, A P ^a   Sims, J J ^a   Markel, M L ^a   Ujhelyi, M R ^a  

^a MEDICAL COLLEGE OF GEORGIA   (United States)

Author keywords

Cardiac electrophysiology; Glyburide; Ion conductance; Pinacidil; Ventricular defibrillation; Ventricular fibrillation

Indexed keywords

ADENOSINE TRIPHOSPHATASE (POTASSIUM); ADENOSINE TRIPHOSPHATASE INHIBITOR; GLIBENCLAMIDE; PINACIDIL; POTASSIUM CHANNEL STIMULATING AGENT;

ACTION POTENTIAL; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DEFIBRILLATION; DRUG ANTAGONISM; DRUG MECHANISM; HEART ELECTROPHYSIOLOGY; HEART REPOLARIZATION; HEART VENTRICLE FIBRILLATION; NONHUMAN; POTASSIUM CONDUCTANCE; POTASSIUM TRANSPORT; PRIORITY JOURNAL; REFRACTORY PERIOD; SWINE;

EID: 0031405957     PISSN: 10742484     EISSN: None     Source Type: Journal    
DOI: 10.1177/107424849700200304     Document Type: Article

References (39)