Programmed cell death (apoptosis) in Alzheimer's disease and Creutzfeldt-Jakob disease

Folia Neuropathologica

Volumn 35, Issue 4, 1997, Pages 233-235

  Jesionek Kupnicka, Dorota ^a   Buczyński, Jarosław ^a   Kordek, Radzisław ^a   Sobów, Tomasz ^a   Kłoszewska, Iwona ^a   Papierz, Wielislaw ^a   Liberski, Paweł P ^a  

^a MEDICAL UNIVERSITY OF LODZ   (Poland)

Author keywords

Alzheimer's disease; Apoptosis; Creutzfeldt Jakob disease

Indexed keywords

AMYLOID BETA PROTEIN; DNA FRAGMENT; NERVE PROTEIN; PRION PROTEIN;

ALZHEIMER DISEASE; ANIMAL; APOPTOSIS; ARTICLE; BRAIN; BRAIN LEVEL; CREUTZFELDT JAKOB DISEASE; HUMAN; METABOLISM; MOUSE; NICK END LABELING; PATHOLOGY;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANIMALS; APOPTOSIS; BRAIN; BRAIN CHEMISTRY; CREUTZFELDT-JAKOB SYNDROME; DNA FRAGMENTATION; HUMANS; IN SITU NICK-END LABELING; MICE; NERVE TISSUE PROTEINS; PRPC PROTEINS;

EID: 0031292557     PISSN: 00283894     EISSN: 1509572X     Source Type: Journal    
DOI: None     Document Type: Article

References (5)

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4. Paradis E, Douillard H, Koutroumanis M, Goodyer C, Le Blanc A: Amyloid β peptide of Alzheimer's disease down-regulates bcl-2 and upregulates bax expression in human neurons. J Neuroscience, 1996, 16, 7533-7539.
5. Su JH, Anderson AJ, Cotman CW: Up-regulation of Bcl-2 is associated with neuronal DNA damage in Alzheimer disease. Neuroreport, 1996, 7, 437-440.