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Volumn 62, Issue 11, 1997, Pages 1306-1310
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Critical telomere shortening regulated by the ataxia-telangiectasia gene acts as a DNA damage signal leading to activation of p53 protein and limited life-span of human diploid fibroblasts. A review
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Author keywords
Aging; ATM; DNA damage; Fibroblasts; P21; P53; Senescence; Telomere
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Indexed keywords
ATAXIA;
ATAXIA TELANGIECTASIA;
PROTEIN P53;
ATAXIA TELANGIECTASIA;
CELL AGING;
CELL CYCLE;
CELL LINE;
CYTOLOGY;
DIPLOIDY;
DNA DAMAGE;
FIBROBLAST;
GENETICS;
HUMAN;
METABOLISM;
MITOSIS;
PHYSIOLOGY;
REVIEW;
SIGNAL TRANSDUCTION;
TELOMERE;
ATAXIA TELANGIECTASIA;
CELL AGING;
CELL CYCLE;
CELL LINE, TRANSFORMED;
DIPLOIDY;
DNA DAMAGE;
FIBROBLASTS;
HUMANS;
MITOSIS;
SIGNAL TRANSDUCTION;
TELOMERE;
TUMOR SUPPRESSOR PROTEIN P53;
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EID: 0031266691
PISSN: 00062979
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
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Times cited : (37)
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References (5)
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