Prevention of kainic acid-induced limbic seizures and fos expression by the GABA-A receptor agonist muscimol

European Journal of Neuroscience

Volumn 9, Issue 1, 1997, Pages 29-40

  Ju, Gong ^a  

^a Institute of Neurosciences   (China)

Author keywords

c fos gene; Immunohistochemistry; Rat brain; Temporal lobe epilepsy

Indexed keywords

4 AMINOBUTYRIC ACID A RECEPTOR STIMULATING AGENT; KAINIC ACID; MUSCIMOL;

AMYGDALOID NUCLEUS; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; CLAUSTRUM; COMPLEX PARTIAL SEIZURE; CONTROLLED STUDY; DENTATE GYRUS; FOREBRAIN; GLOBUS PALLIDUS; HABENULA; HIPPOCAMPUS; MALE; NONHUMAN; NUCLEUS ACCUMBENS; ONCOGENE C FOS; PRIORITY JOURNAL; PUTAMEN; PYRAMIDAL NERVE CELL; RAT; SEPTUM NUCLEUS; THALAMUS NUCLEUS;

ANIMALS; BEHAVIOR, ANIMAL; BRAIN CHEMISTRY; EPILEPSY, TEMPORAL LOBE; EXCITATORY AMINO ACID AGONISTS; GABA AGONISTS; IMMUNOHISTOCHEMISTRY; KAINIC ACID; LIMBIC SYSTEM; MALE; MUSCIMOL; PROTO-ONCOGENE PROTEINS C-FOS; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, GABA-A;

EID: 0031051412     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1460-9568.1997.tb01350.x     Document Type: Article

References (4)

1. Arias, C., Montiel, T. and Tapia, R. (1990) Transmitter release in hippocampal slices from rats with limbic seizures produced by systemic administration of kainic acid. Neurochem. Res., 15, 641-645.
2. Ashwood, T. J. and Wheal, H. V. (1986) Loss of inhibition in CA1 region of kainic acid lesioned hippocampus is not associated with changes in postsynaptic response to GABA. Brain Res., 367, 390-394.
3. Ault, B., Gruenthal, M., Armstrong, D. R. and Nadler, J. V. (1986) Efficacy of baclofen and phenobarbital against the kainic acid limbic seizure-brain damage syndrome. J. Pharmacol. Exp. Ther., 239, 612-617.
4. Baran, H., Lassmann, H. and Homykeiwicz, O. (1988) Behavior and neurochemical changes 6 months after kainic acid. Soc. Neurosci. Abstr., 14, 472.