Microfibrosis produces electrical load variations due to loss of side- to-side cell connections: A major mechanism of structural heart disease arrhythmias

PACE - Pacing and Clinical Electrophysiology

Volumn 20, Issue 2 II, 1997, Pages 397-413

  Spach, Madison S ^a   Boineau, John P ^b  

^a DUKE UNIVERSITY MEDICAL CENTER   (United States)

^b NONE

Author keywords

arrhythmias; microfibrosis; myocardial microstructure; structural heart disease

Indexed keywords

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL JUNCTION; CELL MEMBRANE DEPOLARIZATION; CONTROLLED STUDY; DOG; HEART ARRHYTHMIA; HEART CONDUCTION; HEART DISEASE; HEART ELECTROPHYSIOLOGY; NONHUMAN; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM; STOCHASTIC MODEL;

ANIMALS; ARRHYTHMIA; ATRIAL FUNCTION; CORONARY DISEASE; ELECTROPHYSIOLOGY; FIBROSIS; GAP JUNCTIONS; HEART CONDUCTION SYSTEM; HUMANS; INTERCELLULAR JUNCTIONS; MYOCARDIUM;

EID: 0031049517     PISSN: 01478389     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1540-8159.1997.tb06199.x     Document Type: Article

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