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Volumn 14, Issue 5, 1997, Pages 603-609
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Point mutations can inactivate in vitro and in vivo activities of p16(INK4a)/CDKN2A in human glioma
a,c a a a,b a,b |
Author keywords
CDKN2A; Glioma; Mutation; p16(INK4a); RB; Tumor suppressor gene
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Indexed keywords
CYCLIN DEPENDENT KINASE INHIBITOR;
RETINOBLASTOMA PROTEIN;
ANIMAL CELL;
ARTICLE;
CHROMOSOME 9P;
CONTROLLED STUDY;
GENE REPRESSION;
GLIOMA;
GLIOMA CELL;
GROWTH INHIBITION;
HUMAN;
HUMAN CELL;
MITOSIS INHIBITION;
NONHUMAN;
POINT MUTATION;
PRIORITY JOURNAL;
PROTEIN PHOSPHORYLATION;
TUMOR SUPPRESSOR GENE;
ANIMALIA;
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EID: 0031047550
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1200870 Document Type: Article |
Times cited : (35)
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References (45)
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