Is post-hypoxic-ischemic cell damage associated with excessive ATP consumption rather than a failure of ATP production?

Acta Paediatrica, International Journal of Paediatrics

Volumn 86, Issue 1, 1997, Pages 1-5

  Harkness, R A ^a,b  

^a UNIVERSITY OF OSLO   (Norway)

^b UNIVERSITY OF EDINBURGH   (United Kingdom)

Author keywords

ATP depletion; heat shock proteins; hypoxanthine; hypoxia; inflammation; ischemia; oxidation; protein synthesis and proteolysis; receptors; secondary cell damage mechanisms

Indexed keywords

ADENOSINE TRIPHOSPHATE; HEAT SHOCK PROTEIN;

APOPTOSIS; CELL DAMAGE; CEREBRAL PALSY; ENERGY CONSUMPTION; HUMAN; HYPOXIA; ISCHEMIA; NEWBORN; PRIORITY JOURNAL; PROTEIN SYNTHESIS; REVIEW;

EID: 0031034724     PISSN: 08035253     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1651-2227.1997.tb08820.x     Document Type: Review

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