Azimilide (NE-10064) can prolong or shorten the action potential duration in canine ventricular myocytes: Dependence on blockade of K, Ca, and Na channels

Journal of Cardiovascular Electrophysiology

Volumn 8, Issue 2, 1997, Pages 184-198

  Yao, J A ^a   Tseng, G N ^a  

^a Och Spine at New York Presbyterian Hospitals   (United States)

Author keywords

azimilide; Class III antiarrhythmic agent; inward rectifier current; L type Ca current; rapid delayed rectifier current; slow delayed rectifier current; slowly inactivating Na current; transient outward current

Indexed keywords

AZIMILIDE; CALCIUM CHANNEL; POTASSIUM CHANNEL; SODIUM CHANNEL;

ANIMAL CELL; ANTIARRHYTHMIC ACTIVITY; ARTICLE; DOG; DRUG EFFECT; DRUG POTENCY; HEART ARRHYTHMIA; HEART MUSCLE CELL; HEART MUSCLE POTENTIAL; NONHUMAN; PRIORITY JOURNAL;

EID: 0031017355     PISSN: 10453873     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1540-8167.1997.tb00780.x     Document Type: Article

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