Lymphocyte recruitment following spinal cord injury in mice is altered by prior viral exposure

European Journal of Neuroscience

Volumn 9, Issue 5, 1997, Pages 1000-1007

  Schnell, Lisa ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

CNS lesion; Coronavirus; Immune reaction; T cell response

Indexed keywords

CD4 ANTIGEN; CD8 ANTIGEN; CYTOKINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIBODY TITER; ARTICLE; B LYMPHOCYTE; CELL INFILTRATION; CONTROLLED STUDY; DEMYELINATION; IMMUNOHISTOCHEMISTRY; INFLAMMATION; LYMPHOCYTE; MACROPHAGE; MOUSE; MURINE HEPATITIS CORONAVIRUS; NEUTROPHIL; NONHUMAN; PRIORITY JOURNAL; RAT; SCAR FORMATION; SPINAL CORD INJURY; T LYMPHOCYTE; T LYMPHOCYTE SUBPOPULATION; VIRUS;

ANIMALS; HEPATITIS VIRUSES; IMMUNOHISTOCHEMISTRY; LYMPHOCYTES; MICE; MICE, INBRED C57BL; SPINAL CORD INJURIES;

EID: 0031008893     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1460-9568.1997.tb01450.x     Document Type: Article

References (3)

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2. Bartholdi, D. and Schwab, M. E. (1995) Methylprednisolone inhibits early inflammatory processes but not ischemi cell death after experimental spinal cord lesion in the rat. Brain Res., 672, 177-186.
3. Bell, M. D. and Perry, V. H. (1995) Adhesion molecule expression on murine cerebral endothelium following the injection of a proinflammagen or during acute neuronal degeneration. J. Neurocytol., 24, 695-710.