2-Iminothiazolidine-4-carboxylic acid produces hippocampal CA1 lesions independent of seizure excitation and glutamate receptor activation

NeuroToxicology

Volumn 18, Issue 1, 1997, Pages 191-200

  Bitner, R S ^a   Yim, G K W ^a   Isom, G E ^a  

^a ABBOTT LABORATORIES   (United States)

Author keywords

2 Iminothiazolidine 4 carboxylic Acid; Excitotoxicity; Glutamate receptors; Hippocampal Lesions; Neurotoxicity; Seizures

Indexed keywords

6 CYANO 7 NITRO 2,3 QUINOXALINEDIONE; DIZOCILPINE; EXCITOTOXIN; GLUTAMATE RECEPTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; HIPPOCAMPUS; HISTOLOGY; MALE; MOUSE; NERVE DEGENERATION; NEUROTOXICITY; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL;

6-CYANO-7-NITROQUINOXALINE-2,3-DIONE; ANIMALS; BRAIN ISCHEMIA; DIZOCILPINE MALEATE; EXCITATORY AMINO ACID AGONISTS; GLUTAMIC ACID; HIPPOCAMPUS; INJECTIONS, INTRAVENTRICULAR; MALE; NEUROPROTECTIVE AGENTS; NEUROTOXINS; PROLINE; RATS; RATS, SPRAGUE-DAWLEY; SEIZURES;

EID: 0031001192     PISSN: 0161813X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (32)