On the unique profile of action of clozapine as assessed with fos-protein induction in rat brain regions

Acta Neuropsychiatrica

Volumn 9, Issue 2, 1997, Pages 55-57

  Korf, J ^a   Andries, D ^b   Sebens, J B ^c  

^a UNIVERSITY MEDICAL CENTER GRONINGEN   (Netherlands)

^b UNIVERSITY OF ANTWERP   (Belgium)

^c NONE

Author keywords

clozapine; cross tolerance; fos protein; H1 antagonists

Indexed keywords

CLOZAPINE; DIZOCILPINE; DOPAMINE; GLUTAMIC ACID; HALOPERIDOL; MEPYRAMINE; PROMETHAZINE; PROTEIN;

AMYGDALOID NUCLEUS; ANIMAL EXPERIMENT; BRAIN; CONFERENCE PAPER; DRUG TOLERANCE; HEART RATE; HYPONATREMIA; INTRAPERITONEAL DRUG ADMINISTRATION; NONHUMAN; RAT; SCHIZOPHRENIA;

EID: 0030983639     PISSN: 09242708     EISSN: None     Source Type: Journal    
DOI: 10.1017/S0924270800036796     Document Type: Conference Paper

References (3)

1. Robertson GS, Fibiger HC. Neuroleptics increase c-fos expression in the forebrain: contrasting effects of haloperidol and clozapine. Neuroscience 1992; 46: 315.
2. Sebens JB, Koch T, Ter Horst GJ, et al. Differential Fos-protein induction in rat forebrain regions after acute and long-term haloperidol and clozapine treatment. Eur J Pharmacol 1995; 273: 175.
3. Sebens JB, Koch T, Korf J. Lack of cross-tolerance between haloperidol and clozapine towards Fos-protein induction in rat forebrain regions. Eur J Pharmacol, 1996; 315: 269-75.