Gap junctions promote the bystander effect of herpes simplex virus thymidine kinase in vivo

Cancer Research

Volumn 57, Issue 8, 1997, Pages 1523-1528

  Dilber, M Siraç ^a,b   Abedi, Mohammad R ^a   Christensson, Birger ^a   Bj̈rkstrand, Bo ^a   Kidder, Gerald M ^c   Naus, Christian C G ^c   Gahrton, Gösta ^a   Smith, C I Edvard ^a,b  

^a KAROLINSKA UNIVERSITY HOSPITAL   (Sweden)

^b KAROLINSKA INSTITUTE   (Sweden)

^c UNIVERSITY OF WESTERN ONTARIO   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

CONNEXIN 43; GANCICLOVIR; GAP JUNCTION PROTEIN; SODIUM CHLORIDE; THYMIDINE KINASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL COMMUNICATION; CELL JUNCTION; CONTROLLED STUDY; GENE TRANSFER; GENETIC TRANSDUCTION; GLIOMA CELL; HERPES SIMPLEX VIRUS; INOCULATION; INTRAPERITONEAL DRUG ADMINISTRATION; NONHUMAN; PRIORITY JOURNAL; RAT; SCID MOUSE; SUBCUTANEOUS DRUG ADMINISTRATION; TUMOR REGRESSION; VIRUS GENE;

ANIMALS; ANTIVIRAL AGENTS; BRAIN NEOPLASMS; CELL DIVISION; GANCICLOVIR; GAP JUNCTIONS; GENES, VIRAL; GLIOMA; MICE; MICE, SCID; RATS; THYMIDINE KINASE; TRANSFECTION; TUMOR CELLS, CULTURED;

EID: 0030937485     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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