The scid defect results in much slower repair of DNA double-strand breaks but not high levels of residual breaks

Radiation Research

Volumn 147, Issue 5, 1997, Pages 535-540

  Nevaldine, Barbara ^a   Longo, John A ^a   Hahn, Peter J ^a  

^a SUNY UPSTATE MEDICAL UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DOUBLE STRANDED DNA; PROTEIN KINASE;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; COMBINED IMMUNODEFICIENCY; CONFORMATIONAL TRANSITION; DNA REPAIR; DNA STRAND BREAKAGE; DOSE RESPONSE; ENZYME CONFORMATION; IONIZING RADIATION; MOUSE; MUTAGENESIS; NONHUMAN; PRIORITY JOURNAL; RADIATION DOSE; RADIATION INJURY; SOUTHERN BLOTTING; STRUCTURE ACTIVITY RELATION;

EID: 0030926490     PISSN: 00337587     EISSN: None     Source Type: Journal    
DOI: 10.2307/3579619     Document Type: Article

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