B-Myb function can be markedly enhanced by cyclin A-dependent kinase and protein truncation

Oncogene

Volumn 14, Issue 20, 1997, Pages 2445-2453

  Lane, Sarah ^a   Farlie, Peter ^a   Watson, Roger ^a  

^a IMPERIAL COLLEGE SCHOOL OF MEDICINE   (United Kingdom)

Author keywords

B Myb; Cell cycle; Cyclin A; Transactivation

Indexed keywords

CYCLIN A; PHOSPHOTRANSFERASE; TRANSCRIPTION FACTOR E2F;

ARTICLE; CARBOXY TERMINAL SEQUENCE; CELL CYCLE S PHASE; CELL STRAIN 3T3; GENETIC TRANSCRIPTION; HUMAN; HUMAN CELL; ONCOGENE; PHOSPHORYLATION; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN PROCESSING; TRANSACTIVATION;

3T3 CELLS; ANIMALS; CELL CYCLE; CELL CYCLE PROTEINS; CONSERVED SEQUENCE; DNA-BINDING PROTEINS; HUMANS; MICE; PHOSPHORYLATION; PROTEIN KINASES; TRANS-ACTIVATION (GENETICS); TRANS-ACTIVATORS; TRANSCRIPTION FACTORS; TRANSFECTION; TUMOR CELLS, CULTURED;

EID: 0030919599     PISSN: 09509232     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.onc.1201086     Document Type: Article

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