Susceptibility of cerebellar granule neurons derived from Bcl-2-deficient and transgenic mice to cell death

European Journal of Neuroscience

Volumn 9, Issue 4, 1997, Pages 848-856

  Tsujimoto, Yoshihide ^a  

^a OSAKA UNIVERSITY MEDICAL SCHOOL   (Japan)

Author keywords

A23187; Calcium; Etoposide; Mouse cerebellum; Nimodipine

Indexed keywords

CALCIMYCIN; ETOPOSIDE; NIMODIPINE; PROTEIN BCL 2;

ANIMAL CELL; ARTICLE; BRAIN NERVE CELL; CELL DEATH; CELL SURVIVAL; CEREBELLUM; MOUSE; NONHUMAN; PRIORITY JOURNAL; TRANSGENIC MOUSE;

EID: 0030888097     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1460-9568.1997.tb01434.x     Document Type: Article

References (3)

1. Allsopp, T. E., Wyatt, S., Paterson, H. F. and Davies, A. M. (1993) The protooncogene bcl-2 can selectively rescue neurotrophic factor-dependent neurons from apoptosis. Cell, 73, 295-307.
2. Allsopp, T. E., Kiselev, S., Wyatt, S. and Davies, A. M. (1995) Role of Bcl-2 in the brain-derived neurotrophic factor survival response. Eur. J. Neurosci., 1, 1266-1272.
3. Ankarcrona, M., Dypbukt, J. M., Bonfoco, E., Zhivotovsky, B., Orrenius, S., Lipton, S. A. and Nicotera, P. (1995) Glutamate-induced neuronal death: a succession of necrosis or apoptosis depending on mitochondrial function. Neuron, 15, 961-973.